Fas and perform contribute to the pathogenesis of murine cerebral malaria

Redox Report

Volumn 4, Issue 6, 1999, Pages 334-335

  Potter, S ^a   Chaudhri, G ^a   Hansen, A ^a   Hunt, N H ^a  

^a UNIVERSITY OF SYDNEY   (Australia)

Author keywords

[No Author keywords available]

Indexed keywords

EVANS BLUE; FAS ANTIGEN; PERFORIN;

ANEMIA; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BLOOD BRAIN BARRIER; BRAIN EDEMA; BRAIN MALARIA; CELL ADHESION; COMA; CONTROLLED STUDY; DISEASE SEVERITY; HEMIPLEGIA; HISTOPATHOLOGY; MONOCYTE; MOUSE; NONHUMAN; PARASITEMIA; PATHOGENESIS; PETECHIA; PRIORITY JOURNAL; PROTEIN EXPRESSION; SYMPTOM;

ANIMALIA; MURINAE;

EID: 33745480947     PISSN: 13510002     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (6)

1. Yanez DM, Manning DD, Cooley AJ, Weidanz WP, van der Heyde HC. Participation of lymphocyte subpopulations in the pathogenesis of experimental murine malaria. J Immunol 1996; 157: 1620-1624.
2. Spiclman J, Lee RK, Podack ER. Perforin/Fas-ligand double deficiency is associated with macrophage expansion and severe pancreatitis. J Immimol 1998; 161: 7063-7070.
3. Medana IM, Hunt NH, Chan-Ling T. Early activation of microglia in the pathogenesis of murine cerebral malaria. Glia 1997; 19: 91-103.
4. Choi C, Park J Y, Lee J et al. Fas ligand and Fas are expressed constitutively in human astrocytes and the expression increases with IL-1, IL-6, TNF-α or IFN-γ. J Immunol 1999; 162: 1889-1895.
5. Saas P, Boucraut J, Quiquerez A-L et al. CD95 (Fas/Apo-1) as a receptor governing astrocyte apoptotic or inflammatory responses: a key role in brain inflammation? J Iimmmol 1999; 162: 2326-2333.
6. Bonetti B, Pohl J, Gao Y-L, Raine C. Cell death during autoimmune demyelination: effector but not target cells are eliminated by apoplosis. J Immimol 1997; 159: 5733-5741.