MafK overexpression in pancreatic β-cells caused impairment of glucose-stimulated insulin secretion

Biochemical and Biophysical Research Communications

Volumn 346, Issue 3, 2006, Pages 671-680

  Shimohata, Homare ^a   Yoh, Keigyou ^b   Morito, Naoki ^a   Shimano, Hitoshi ^c   Kudo, Takashi ^a   Takahashi, Satoru ^a  

^a Graduate School of Comprehensive Human Sciences   (Japan)

^b UNIVERSITY OF TSUKUBA   (Japan)

^c UNIVERSITY OF TSUKUBA   (Japan)

Author keywords

Diabetes mellitus; Insulin; Maf; Transcription factor; Transgenic mouse

Indexed keywords

DNA; GLUCOSE; INSULIN; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR MAFA; TRANSCRIPTION FACTOR MAFK; UNCLASSIFIED DRUG;

ADULTHOOD; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CELL FUNCTION; CELL SIZE; CONTROLLED STUDY; DNA BINDING; DOWN REGULATION; GEL MOBILITY SHIFT ASSAY; GENE OVEREXPRESSION; GLUCOSE TOLERANCE TEST; GROUPS BY AGE; HISTOLOGY; HYPERGLYCEMIA; HYPERTROPHY; INSULIN RELEASE; MALE; MOUSE; NONHUMAN; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; PROTEIN FUNCTION; TRANSGENIC MOUSE;

AGING; ANIMALS; BODY WEIGHT; CELL SHAPE; GENE EXPRESSION REGULATION; GLUCOSE; HYPERGLYCEMIA; INSULIN; INSULIN-SECRETING CELLS; MAF TRANSCRIPTION FACTORS, LARGE; MAFK TRANSCRIPTION FACTOR; MICE; MICE, TRANSGENIC; NIH 3T3 CELLS; PROMOTER REGIONS (GENETICS); PROTEIN BINDING; RESPONSE ELEMENTS;

MUS MUSCULUS;

EID: 33745197298     PISSN: 0006291X     EISSN: 10902104     Source Type: Journal    
DOI: 10.1016/j.bbrc.2006.05.184     Document Type: Article

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