Novel β subunit mutation causes a slow-channel syndrome by enhancing activation and decreasing the rate of agonist dissociation

Molecular and Cellular Neuroscience

Volumn 32, Issue 1-2, 2006, Pages 82-90

  Navedo, Manuel F ^a   Lasalde Dominicci, José A ^b   Báez Pagán, Carlos A ^b   Díaz Pérez, Luzed ^b   Rojas, Legier V ^c   Maselli, Ricardo A ^d   Staub, Julie ^e   Schott, Kelly ^e   Zayas, Roberto ^e   Gomez, Christopher M ^e  

^a UNIVERSITY OF WASHINGTON   (United States)

^b UNIVERSITY OF PUERTO RICO   (Puerto Rico)

^c UNIVERSIDAD CENTRAL DEL CARIBE   (United States)

^d UNIVERSITY OF CALIFORNIA   (United States)

^e UNIVERSITY OF MINNESOTA   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CHOLINERGIC RECEPTOR; CHOLINERGIC RECEPTOR BETA SUBUNIT; RECEPTOR SUBUNIT; UNCLASSIFIED DRUG;

ADRENERGIC TRANSMISSION; AGED; AMINO ACID SUBSTITUTION; ANIMAL CELL; ARTICLE; CALCIUM TRANSPORT; CONGENITAL MYASTHENIC SYNDROME; CONTROLLED STUDY; DISSOCIATION; ELECTRIC CURRENT; EMBRYO; GENE MUTATION; HUMAN; HUMAN CELL; KINETICS; MALE; MUSCLE ACTION POTENTIAL; MUSCLE WEAKNESS; MUTATIONAL ANALYSIS; NONHUMAN; OOCYTE; PRIORITY JOURNAL; SYNAPSE; WILD TYPE; XENOPUS;

ACETYLCHOLINE; AGED; AMINO ACID SUBSTITUTION; ANIMALS; CALCIUM SIGNALING; CELL MEMBRANE PERMEABILITY; EXCITATORY POSTSYNAPTIC POTENTIALS; FEMALE; HUMANS; ION CHANNELS; KINETICS; MALE; MUSCLE, SKELETAL; MUTATION; MYASTHENIC SYNDROMES, CONGENITAL; NEUROMUSCULAR JUNCTION; OOCYTES; PEDIGREE; RECEPTORS, NICOTINIC; SYNAPTIC TRANSMISSION; XENOPUS LAEVIS;

EID: 33744970023     PISSN: 10447431     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.mcn.2006.02.004     Document Type: Article

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