Can promotion of initiated cells be explained by excess replacement of radiation-inactivated neighbor cells?

Radiation Research

Volumn 165, Issue 6, 2006, Pages 741-744

  Bijwaard, Harmen ^a   Brugmans, Marco J P ^a   Schöllnberger, Helmut ^b  

^a NATIONAL INSTITUTE FOR PUBLIC HEALTH AND THE ENVIRONMENT   (Netherlands)

^b UNIVERSITY OF SALZBURG   (Austria)

Author keywords

[No Author keywords available]

Indexed keywords

ARTICLE; CELL ACTIVATION; CELL CYCLE; IRRADIATION; PRIORITY JOURNAL; QUANTITATIVE ANALYSIS; RADIATION EXPOSURE;

BYSTANDER EFFECT; CELL SURVIVAL; CELL TRANSFORMATION, NEOPLASTIC; COMPUTER SIMULATION; DOSE-RESPONSE RELATIONSHIP, RADIATION; MODELS, BIOLOGICAL; NEOPLASMS; NEOPLASMS, RADIATION-INDUCED; RADIATION DOSAGE;

EID: 33744806917     PISSN: 00337587     EISSN: None     Source Type: Journal    
DOI: 10.1667/RR3548.1     Document Type: Article

References (4)

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3. H. Steinhaus, Mathematical Snapshots, 3rd ed., p. 202. Dover, New York, 1999.
4. S. H. Moolgavkar, E. G. Luebeck, A. Buchmann and K. W. Bock, Quantitative analysis of enzyme-altered liver foci in rats initiated with diethylnitrosamine and promoted with 2,3,7,8-tetrachlorodibenzo-p-dioxin or 1,2,3,4,6,7,8- heptachlorodibenzo-p-dioxin. Toxicol. Appl. Pharmacol. 138, 31-42 (1996).