-
1
-
-
0014289538
-
Studies on blood capillaries. I. General organization of blood capillaries in muscle
-
Bruns, R.R., and Palade, G.E. 1968. Studies on blood capillaries. I. General organization of blood capillaries in muscle. J. Cell Biol. 37:244-276.
-
(1968)
J. Cell Biol.
, vol.37
, pp. 244-276
-
-
Bruns, R.R.1
Palade, G.E.2
-
2
-
-
0035964954
-
Loss of caveolae, vascular dysfunction, and pulmonary defects in caveolin-1 gene-disrupted mice
-
Drab, M., et al. 2001. Loss of caveolae, vascular dysfunction, and pulmonary defects in caveolin-1 gene-disrupted mice. Science. 293:2449-2452.
-
(2001)
Science
, vol.293
, pp. 2449-2452
-
-
Drab, M.1
-
3
-
-
0035851197
-
Caveolin-1 null mice are viable but show evidence of hyperproliferative and vascular abnormalities
-
Razani, B., et al. 2001. Caveolin-1 null mice are viable but show evidence of hyperproliferative and vascular abnormalities. J. Biol. Chem. 276:38121-38138.
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 38121-38138
-
-
Razani, B.1
-
4
-
-
0032723268
-
Caveolins, liquid-ordered domains, and signal transduction
-
Smart, E.J., et al. 1999. Caveolins, liquid-ordered domains, and signal transduction. Mol. Cell. Biol. 19:7289-7304.
-
(1999)
Mol. Cell. Biol.
, vol.19
, pp. 7289-7304
-
-
Smart, E.J.1
-
5
-
-
2942685631
-
Caveolae and caveolins in the cardiovascular system
-
Gratton, J.P., Bernatchez, P., and Sessa, W.C. 2004. Caveolae and caveolins in the cardiovascular system. Circ. Res. 94:1408-1417.
-
(2004)
Circ. Res.
, vol.94
, pp. 1408-1417
-
-
Gratton, J.P.1
Bernatchez, P.2
Sessa, W.C.3
-
6
-
-
0028925762
-
Caveolae from luminal plasmalemma of rat lung endothelium: Microdomains enriched in caveolin, Ca(2+)-ATPase, and inositol trisphosphate receptor
-
Schnitzer, J.E., Oh, P., Jacobson, B.S., and Dvorak, A.M. 1995. Caveolae from luminal plasmalemma of rat lung endothelium: microdomains enriched in caveolin, Ca(2+)-ATPase, and inositol trisphosphate receptor. Proc. Natl. Acad. Sci. U. S. A. 92:1759-1763.
-
(1995)
Proc. Natl. Acad. Sci. U. S. A.
, vol.92
, pp. 1759-1763
-
-
Schnitzer, J.E.1
Oh, P.2
Jacobson, B.S.3
Dvorak, A.M.4
-
7
-
-
0032567415
-
In situ flow activates endothelial nitric oxide synthase in luminal caveolae of endothelium with rapid caveolin dissociation and calmodulin association
-
Rizzo, V., McIntosh, D.P., Oh, P., and Schnitzer, J.E. 1998. In situ flow activates endothelial nitric oxide synthase in luminal caveolae of endothelium with rapid caveolin dissociation and calmodulin association. J. Biol. Chem. 273:34724-34729.
-
(1998)
J. Biol. Chem.
, vol.273
, pp. 34724-34729
-
-
Rizzo, V.1
McIntosh, D.P.2
Oh, P.3
Schnitzer, J.E.4
-
8
-
-
0032500605
-
Rapid mechanotransduction in situ at the luminal cell surface of vascular endothelium and its caveolae
-
Rizzo, V., Sung, A., Oh, P., and Schnitzer, J.E. 1998. Rapid mechanotransduction in situ at the luminal cell surface of vascular endothelium and its caveolae. J. Biol. Chem. 273:26323-26329.
-
(1998)
J. Biol. Chem.
, vol.273
, pp. 26323-26329
-
-
Rizzo, V.1
Sung, A.2
Oh, P.3
Schnitzer, J.E.4
-
9
-
-
0041357203
-
Chronic shear induces caveolae formation and alters ERK and Akt responses in endothelial cells
-
Boyd, N.L., et al. 2003. Chronic shear induces caveolae formation and alters ERK and Akt responses in endothelial cells. Am. J. Physiol. Heart Circ. Physiol. 285:H1113-H1122.
-
(2003)
Am. J. Physiol. Heart Circ. Physiol.
, vol.285
-
-
Boyd, N.L.1
-
10
-
-
0141676760
-
Recruitment of endothelial caveolae into mechanotransduction pathways by flow conditioning in vitro
-
Rizzo, V., Morton, C., DePaola, N., Schnitzer, J.E., and Davies, P.F. 2003. Recruitment of endothelial caveolae into mechanotransduction pathways by flow conditioning in vitro. Am. J. Physiol. Heart Circ. Physiol. 285:H1720-H1729.
-
(2003)
Am. J. Physiol. Heart Circ. Physiol.
, vol.285
-
-
Rizzo, V.1
Morton, C.2
DePaola, N.3
Schnitzer, J.E.4
Davies, P.F.5
-
11
-
-
0036179393
-
Shear stress induces caveolin-1 translocation in cultured endothelial cells
-
Sun, R.J., Muller, S., Stoltz, J.F., and Wang, X. 2002. Shear stress induces caveolin-1 translocation in cultured endothelial cells. Eur. Biophys. J. 30:605-611.
-
(2002)
Eur. Biophys. J.
, vol.30
, pp. 605-611
-
-
Sun, R.J.1
Muller, S.2
Stoltz, J.F.3
Wang, X.4
-
12
-
-
0036975880
-
Caveolin-1 redistribution in human endothelial cells induced by laminar flow and cytokine
-
Sun, R.J., Muller, S., Zhuang, F.Y., Stoltz, J.F., and Wang, X. 2003. Caveolin-1 redistribution in human endothelial cells induced by laminar flow and cytokine. Biorheology. 40:31-39.
-
(2003)
Biorheology
, vol.40
, pp. 31-39
-
-
Sun, R.J.1
Muller, S.2
Zhuang, F.Y.3
Stoltz, J.F.4
Wang, X.5
-
13
-
-
0033610896
-
Plasma membrane cholesterol is a key molecule in shear stress-dependent activation of extracellular signal-regulated kinase
-
Park, H., et al. 1998. Plasma membrane cholesterol is a key molecule in shear stress-dependent activation of extracellular signal-regulated kinase. J. Biol. Chem. 273:32304-32311.
-
(1998)
J. Biol. Chem.
, vol.273
, pp. 32304-32311
-
-
Park, H.1
-
14
-
-
10344238967
-
Cyclosporin A inhibits flow-mediated activation of endothelial nitricoxide synthase by altering cholesterol content in caveolae
-
Lungu, A.O., et al. 2004. Cyclosporin A inhibits flow-mediated activation of endothelial nitricoxide synthase by altering cholesterol content in caveolae. J. Biol. Chem. 279:48794-48800.
-
(2004)
J. Biol. Chem.
, vol.279
, pp. 48794-48800
-
-
Lungu, A.O.1
-
15
-
-
0344585437
-
Lipid rafts: Elusive or illusive?
-
Munro, S. 2003. Lipid rafts: elusive or illusive? Cell. 115:377-388.
-
(2003)
Cell.
, vol.115
, pp. 377-388
-
-
Munro, S.1
-
16
-
-
0035923728
-
Distinction between signaling mechanisms in lipid rafts vs. caveolae
-
Sowa, G., Pypaert, M., and Sessa, W.C. 2001. Distinction between signaling mechanisms in lipid rafts vs. caveolae. Proc. Natl. Acad. Sci. U. S. A. 98:14072-14077.
-
(2001)
Proc. Natl. Acad. Sci. U. S. A.
, vol.98
, pp. 14072-14077
-
-
Sowa, G.1
Pypaert, M.2
Sessa, W.C.3
-
17
-
-
19944428563
-
Endothelial-specific expression of caveolin-1 impairs microvascular permeability and angiogenesis
-
Bauer, P.M., et al. 2005. Endothelial-specific expression of caveolin-1 impairs microvascular permeability and angiogenesis. Proc. Natl. Acad. Sci. U. S. A. 102:204-209.
-
(2005)
Proc. Natl. Acad. Sci. U. S. A.
, vol.102
, pp. 204-209
-
-
Bauer, P.M.1
-
18
-
-
0033520479
-
Caveolin-2 localizes to the golgi complex but redistributes to plasma membrane, caveolae, and rafts when co-expressed with caveolin-1
-
Mora, R., et al. 1999. Caveolin-2 localizes to the golgi complex but redistributes to plasma membrane, caveolae, and rafts when co-expressed with caveolin-1. J. Biol. Chem. 274:25708-25717.
-
(1999)
J. Biol. Chem.
, vol.274
, pp. 25708-25717
-
-
Mora, R.1
-
19
-
-
0033520374
-
Expression of caveolin-1 is required for the transport of caveolin-2 to the plasma membrane. Retention of caveolin-2 at the level of the golgi complex
-
Parolini, I., et al. 1999. Expression of caveolin-1 is required for the transport of caveolin-2 to the plasma membrane. Retention of caveolin-2 at the level of the golgi complex. J. Biol. Chem. 274:25718-25725.
-
(1999)
J. Biol. Chem.
, vol.274
, pp. 25718-25725
-
-
Parolini, I.1
-
20
-
-
0032520153
-
Direct evidence for the importance of endothelium-derived nitric oxide in vascular remodeling
-
Rudic, R.D., et al. 1998. Direct evidence for the importance of endothelium-derived nitric oxide in vascular remodeling. J. Clin. Invest. 101:731-736.
-
(1998)
J. Clin. Invest.
, vol.101
, pp. 731-736
-
-
Rudic, R.D.1
-
21
-
-
0034625722
-
Temporal events underlying arterial remodeling after chronic flow reduction in mice: Correlation of structural changes with a deficit in basal nitric oxide synthesis
-
Rudic, R.D., Bucci, M., Fulton, D., Segal, S.S., and Sessa, W.C. 2000. Temporal events underlying arterial remodeling after chronic flow reduction in mice: correlation of structural changes with a deficit in basal nitric oxide synthesis. Circ. Res. 86:1160-1166.
-
(2000)
Circ. Res.
, vol.86
, pp. 1160-1166
-
-
Rudic, R.D.1
Bucci, M.2
Fulton, D.3
Segal, S.S.4
Sessa, W.C.5
-
22
-
-
0035970540
-
Decreased flow-dependent dilation in carotid arteries of tissue kallikrein-knockout mice
-
Bergaya, S., et al. 2001. Decreased flow-dependent dilation in carotid arteries of tissue kallikrein-knockout mice. Circ. Res. 88:593-599.
-
(2001)
Circ. Res.
, vol.88
, pp. 593-599
-
-
Bergaya, S.1
-
23
-
-
3042820768
-
Flow-dependent dilation mediated by endogenous kinins requires angiotensin AT2 receptors
-
Bergaya, S., et al. 2004. Flow-dependent dilation mediated by endogenous kinins requires angiotensin AT2 receptors. Circ. Res. 94:1623-1629.
-
(2004)
Circ. Res.
, vol.94
, pp. 1623-1629
-
-
Bergaya, S.1
-
24
-
-
0029824049
-
Components of flow-induced dilation in rat perfused coronary artery
-
Vequaud, P., and Freslon, J.L. 1996. Components of flow-induced dilation in rat perfused coronary artery. Cell Biol. Toxicol. 12:227-232.
-
(1996)
Cell Biol. Toxicol.
, vol.12
, pp. 227-232
-
-
Vequaud, P.1
Freslon, J.L.2
-
25
-
-
0034529950
-
In vivo delivery of the caveolin-1 scaffolding domain inhibits nitric oxide synthesis and reduces inflammation
-
Bucci, M., et al. 2000. In vivo delivery of the caveolin-1 scaffolding domain inhibits nitric oxide synthesis and reduces inflammation. Nat. Med. 6:1362-1367.
-
(2000)
Nat. Med.
, vol.6
, pp. 1362-1367
-
-
Bucci, M.1
-
26
-
-
0037013179
-
Functional reconstitution of endothelial nitric oxide synthase reveals the importance of serine 1179 in endothelium-dependent vasomotion
-
Scotland, R.S., et al. 2002. Functional reconstitution of endothelial nitric oxide synthase reveals the importance of serine 1179 in endothelium-dependent vasomotion. Circ. Res. 90:904-910.
-
(2002)
Circ. Res.
, vol.90
, pp. 904-910
-
-
Scotland, R.S.1
-
27
-
-
0033542476
-
Activation of nitric oxide synthase in endothelial cells by Akt-dependent phosphorylation
-
Dimmeler, S., et al. 1999. Activation of nitric oxide synthase in endothelial cells by Akt-dependent phosphorylation. Nature. 399:601-605.
-
(1999)
Nature
, vol.399
, pp. 601-605
-
-
Dimmeler, S.1
-
28
-
-
0033542414
-
Regulation of endothelium-derived nitric oxide production by the protein kinase Akt
-
Fulton, D., et al. 1999. Regulation of endothelium-derived nitric oxide production by the protein kinase Akt. Nature. 399:597-601.
-
(1999)
Nature
, vol.399
, pp. 597-601
-
-
Fulton, D.1
-
29
-
-
33644694921
-
Gab1, SHP2, and protein kinase A are crucial for the activation of the endothelial NO synthase by fluid shear stress
-
Dixit, M., et al. 2005. Gab1, SHP2, and protein kinase A are crucial for the activation of the endothelial NO synthase by fluid shear stress. Circ. Res. 97:1236-1244.
-
(2005)
Circ. Res.
, vol.97
, pp. 1236-1244
-
-
Dixit, M.1
-
30
-
-
0141648341
-
Functional expression of endothelial nitric oxide synthase fused to green fluorescent protein in transgenic mice
-
van Haperen, R., et al. 2003. Functional expression of endothelial nitric oxide synthase fused to green fluorescent protein in transgenic mice. Am. J. Pathol. 163:1677-1686.
-
(2003)
Am. J. Pathol.
, vol.163
, pp. 1677-1686
-
-
Van Haperen, R.1
-
31
-
-
28444449470
-
Shear stress affects the intracellular distribution of eNOS: Direct demonstration by a novel in vivo technique
-
Cheng, C., et al. 2005. Shear stress affects the intracellular distribution of eNOS: direct demonstration by a novel in vivo technique. Blood. 106:3691-3698.
-
(2005)
Blood
, vol.106
, pp. 3691-3698
-
-
Cheng, C.1
-
32
-
-
0022048404
-
Flow restriction of one carotid artery in juvenile rats inhibits growth of arterial diameter
-
Guyton, J.R., and Hartley, C.J. 1985. Flow restriction of one carotid artery in juvenile rats inhibits growth of arterial diameter. Am. J. Physiol. 248:H540-H546.
-
(1985)
Am. J. Physiol.
, vol.248
-
-
Guyton, J.R.1
Hartley, C.J.2
-
33
-
-
0022587290
-
Reductions in arterial diameter produced by chronic decreases in blood flow are endothelium-dependent
-
Langille, B.L., and O'Donnell, F. 1986. Reductions in arterial diameter produced by chronic decreases in blood flow are endothelium-dependent. Science. 231:405-407.
-
(1986)
Science
, vol.231
, pp. 405-407
-
-
Langille, B.L.1
O'Donnell, F.2
-
34
-
-
3142579790
-
Caveolin-1 deficiency stimulates neointima formation during vascular injury
-
Hassan, G.S., Jasmin, J.F., Schubert, W., Frank, P.G., and Lisanti, M.P. 2004. Caveolin-1 deficiency stimulates neointima formation during vascular injury. Biochemistry. 43:8312-8321.
-
(2004)
Biochemistry
, vol.43
, pp. 8312-8321
-
-
Hassan, G.S.1
Jasmin, J.F.2
Schubert, W.3
Frank, P.G.4
Lisanti, M.P.5
-
35
-
-
3343020739
-
Caveolin-1 expression is critical for vascular endothelial growth factor-induced ischemic hindlimb collateralization and nitric oxide-mediated angiogenesis
-
Sonveaux, P., et al. 2004. Caveolin-1 expression is critical for vascular endothelial growth factor-induced ischemic hindlimb collateralization and nitric oxide-mediated angiogenesis. Circ. Res. 95:154-161.
-
(2004)
Circ. Res.
, vol.95
, pp. 154-161
-
-
Sonveaux, P.1
-
36
-
-
4644261120
-
Small interfering RNA-mediated down-regulation of caveolin-1 differentially modulates signaling pathways in endothelial cells
-
Gonzalez, E., Nagiel, A., Lin, A.J., Golan, D.E., and Michel, T. 2004. Small interfering RNA-mediated down-regulation of caveolin-1 differentially modulates signaling pathways in endothelial cells. J. Biol. Chem. 279:40659-40669.
-
(2004)
J. Biol. Chem.
, vol.279
, pp. 40659-40669
-
-
Gonzalez, E.1
Nagiel, A.2
Lin, A.J.3
Golan, D.E.4
Michel, T.5
-
37
-
-
0029022062
-
The Golgi association of endothelial nitric oxide synthase is necessary for the efficient synthesis of nitric oxide
-
Sessa, W.C., et al. 1995. The Golgi association of endothelial nitric oxide synthase is necessary for the efficient synthesis of nitric oxide. J. Biol. Chem. 270:17641-17644.
-
(1995)
J. Biol. Chem.
, vol.270
, pp. 17641-17644
-
-
Sessa, W.C.1
-
38
-
-
25144510997
-
A mechanosensory complex that mediates the endothelial cell response to fluid shear stress
-
Tzima, E., et al. 2005. A mechanosensory complex that mediates the endothelial cell response to fluid shear stress. Nature. 437:426-431.
-
(2005)
Nature
, vol.437
, pp. 426-431
-
-
Tzima, E.1
-
39
-
-
5344230667
-
PECAM-1 interacts with nitric oxide synthase in human endothelial cells: Implication for flow-induced nitric oxide synthase activation
-
Dusserre, N., et al. 2004. PECAM-1 interacts with nitric oxide synthase in human endothelial cells: implication for flow-induced nitric oxide synthase activation. Arterioscler. Thromb. Vasc. Biol. 24:1796-1802.
-
(2004)
Arterioscler. Thromb. Vasc. Biol.
, vol.24
, pp. 1796-1802
-
-
Dusserre, N.1
-
40
-
-
0037135988
-
Evidence for a role of platelet endothelial cell adhesion molecule-1 in endothelial cell mechanosignal transduction: Is it a mechanoresponsive molecule?
-
Osawa, M., Masuda, M., Kusano, K., and Fujiwara, K. 2002. Evidence for a role of platelet endothelial cell adhesion molecule-1 in endothelial cell mechanosignal transduction:is it a mechanoresponsive molecule? J. Cell Biol. 158:773-785.
-
(2002)
J. Cell Biol.
, vol.158
, pp. 773-785
-
-
Osawa, M.1
Masuda, M.2
Kusano, K.3
Fujiwara, K.4
-
41
-
-
23244440759
-
PECAM-1 mediates NO-dependent dilation of arterioles to high temporal gradients of shear stress
-
Bagi, Z., et al. 2005. PECAM-1 mediates NO-dependent dilation of arterioles to high temporal gradients of shear stress. Arterioscler. Thromb. Vasc. Biol. 25:1590-1595.
-
(2005)
Arterioscler. Thromb. Vasc. Biol.
, vol.25
, pp. 1590-1595
-
-
Bagi, Z.1
-
42
-
-
3142732827
-
Role of lateral cell-cell border location and extracellular/transmembrane domains in PECAM/CD31 mechanosensation
-
Kaufman, D.A., Albelda, S.M., Sun, J., and Davies, P.F. 2004. Role of lateral cell-cell border location and extracellular/transmembrane domains in PECAM/CD31 mechanosensation. Biochem. Biophys. Res. Commun. 320:1076-1081.
-
(2004)
Biochem. Biophys. Res. Commun.
, vol.320
, pp. 1076-1081
-
-
Kaufman, D.A.1
Albelda, S.M.2
Sun, J.3
Davies, P.F.4
-
43
-
-
15744369759
-
MAPKs (ERK1/2, p38) and AKT can be phosphorylated by shear stress independently of platelet endothelial cell adhesion molecule-1 (CD31) in vascular endothelial cells
-
Sumpio, B.E., et al. 2005. MAPKs (ERK1/2, p38) and AKT can be phosphorylated by shear stress independently of platelet endothelial cell adhesion molecule-1 (CD31) in vascular endothelial cells. J. Biol. Chem. 280:11185-11191.
-
(2005)
J. Biol. Chem.
, vol.280
, pp. 11185-11191
-
-
Sumpio, B.E.1
-
44
-
-
0036838172
-
Interplay between integrins and FLK-1 in shear stress-induced signaling
-
Wang, Y., et al. 2002. Interplay between integrins and FLK-1 in shear stress-induced signaling. Am. J. Physiol. Cell Physiol. 283:C1540-C1547.
-
(2002)
Am. J. Physiol. Cell Physiol.
, vol.283
-
-
Wang, Y.1
-
45
-
-
16844381996
-
Flow shear stress stimulates Gab1 tyrosine phosphorylation to mediate protein kinase B and endothelial nitric-oxide synthase activation in endothelial cells
-
Jin, Z.G., Wong, C., Wu, J., and Berk, B.C. 2005. Flow shear stress stimulates Gab1 tyrosine phosphorylation to mediate protein kinase B and endothelial nitric-oxide synthase activation in endothelial cells. J. Biol. Chem. 280:12305-12309.
-
(2005)
J. Biol. Chem.
, vol.280
, pp. 12305-12309
-
-
Jin, Z.G.1
Wong, C.2
Wu, J.3
Berk, B.C.4
-
46
-
-
33644805287
-
Role of focal adhesion kinase in flow-induced dilation of coronary arterioles
-
Koshida, R., et al. 2005. Role of focal adhesion kinase in flow-induced dilation of coronary arterioles. Arterioscler. Thromb. Vasc. Biol. 25:2548-2553.
-
(2005)
Arterioscler. Thromb. Vasc. Biol.
, vol.25
, pp. 2548-2553
-
-
Koshida, R.1
-
47
-
-
0344507516
-
A role for caveolin and the urokinase receptor in integrin-mediated adhesion and signaling
-
Wei, Y., Yang, X., Liu, Q., Wilkins, J.A., and Chapman, H.A. 1999. A role for caveolin and the urokinase receptor in integrin-mediated adhesion and signaling. J. Cell Biol. 144:1285-1294.
-
(1999)
J. Cell Biol.
, vol.144
, pp. 1285-1294
-
-
Wei, Y.1
Yang, X.2
Liu, Q.3
Wilkins, J.A.4
Chapman, H.A.5
-
48
-
-
12344250442
-
Integrin mechanotransduction stimulates caveolin-1 phosphorylation and recruitment of Csk to mediate actin reorganization
-
Radel, C., and Rizzo, V. 2005. Integrin mechanotransduction stimulates caveolin-1 phosphorylation and recruitment of Csk to mediate actin reorganization. Am. J. Physiol. Heart Circ. Physiol. 288:H936-H945.
-
(2005)
Am. J. Physiol. Heart Circ. Physiol.
, vol.288
-
-
Radel, C.1
Rizzo, V.2
|