Lead increases lipopolysaccharide-induced liver injury through tumor necrosis factor-α overexpression by monocytes/macrophages: Role of protien kinase C and p42/44 mitogen-activated protein kinase

Environmental Health Perspectives

Volumn 114, Issue 4, 2006, Pages 507-513

  Cheng, Yu Jung ^a   Chang, Bei Chang ^a   Liu, Ming Yie ^a  

^a NATIONAL CHENG KUNG UNIVERSITY   (Taiwan)

Author keywords

Lead; Lipopolysaccharide; Liver injury; Monocytes macrophages; P42 44 mitogen activated protein kinase; Protein kinace C; Tumor necrosis factor

Indexed keywords

LEAD; LIPOPOLYSACCHARIDE; MITOGEN ACTIVATED PROTEIN KINASE 1; MITOGEN ACTIVATED PROTEIN KINASE 3; PROTEIN KINASE C; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL CULTURE; CONTROLLED STUDY; CYTOKINE PRODUCTION; ENVIRONMENTAL EXPOSURE; ENZYME PHOSPHORYLATION; LIVER FUNCTION; LIVER INJURY; LIVER TOXICITY; MONOCYTE; MOUSE; MOUSE STRAIN; NONHUMAN; PATHOGENESIS; PERITONEUM MACROPHAGE; PRIORITY JOURNAL; PROTEIN BLOOD LEVEL; PROTEIN EXPRESSION; PROTEIN FUNCTION; T LYMPHOCYTE;

ANIMALS; CELL LINE; ENZYME ACTIVATION; LEAD; LIPOPOLYSACCHARIDES; LIVER; MACROPHAGES; MICE; MITOGEN-ACTIVATED PROTEIN KINASE 1; MITOGEN-ACTIVATED PROTEIN KINASE 3; MONOCYTES; PROTEIN KINASE C; TUMOR NECROSIS FACTOR-ALPHA;

ANIMALIA;

EID: 33645651010     PISSN: 00916765     EISSN: 15529924     Source Type: Journal    
DOI: 10.1289/ehp.8550     Document Type: Article

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