Expression of cathepsin L in human tumor cells is under the control of distinct regulatory mechanisms

Oncogene

Volumn 25, Issue 10, 2006, Pages 1474-1484

  Jean, D ^a   Rousselet, N ^a   Frade, R ^a  

^a Parc Scientifique de Luminy   (France)

Author keywords

DNA methylation; Gene amplification; mRNA stability; Transcription regulation

Indexed keywords

CATHEPSIN L; CYSTEINE PROTEINASE; DINUCLEOTIDE; MESSENGER RNA; REGULATOR PROTEIN; TRANSCRIPTION FACTOR; TRANSCRIPTION FACTOR NF Y; TRANSCRIPTION FACTOR SP1; TRANSCRIPTION FACTOR SP2; TRANSCRIPTION FACTOR SP3; UNCLASSIFIED DRUG;

ARTICLE; B CELL LYMPHOMA; CPG ISLAND; DNA METHYLATION; GENE ACTIVITY; GENE AMPLIFICATION; GENE EXPRESSION REGULATION; GENE LOCATION; GENE OVEREXPRESSION; GENE SILENCING; HUMAN; HUMAN CELL; LYMPHOMA CELL LINE; MELANOMA; MELANOMA CELL; PRIORITY JOURNAL; PROMOTER REGION; PROTEIN EXPRESSION; REGULATORY MECHANISM; RNA ANALYSIS; RNA STABILITY; TRANSCRIPTION REGULATION; TUMOR CELL;

ANIMALS; BASE SEQUENCE; CATHEPSINS; CCAAT-BINDING FACTOR; CELL LINE, TUMOR; CYSTEINE ENDOPEPTIDASES; DNA METHYLATION; DNA, NEOPLASM; GENE EXPRESSION REGULATION, NEOPLASTIC; HUMANS; LYMPHOMA; MICE; MICE, NUDE; MOLECULAR SEQUENCE DATA; NEOPLASM PROTEINS; PROMOTER REGIONS (GENETICS); PROTEIN BINDING; RNA STABILITY; RNA, MESSENGER; SP TRANSCRIPTION FACTORS;

EID: 33644833718     PISSN: 09509232     EISSN: 14765594     Source Type: Journal    
DOI: 10.1038/sj.onc.1209196     Document Type: Article

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