CFTR is required for PKA-regulated ATP sensitivity of Kir1.1 potassium channels in mouse kidney

Journal of Clinical Investigation

Volumn 116, Issue 3, 2006, Pages 797-807

  Lu, Ming ^a   Leng, Qiang ^a   Egan, Marie E ^a   Caplan, Michael J ^a   Boulpaep, Emile L ^a   Giebisch, Gerhard H ^a   Hebert, Steven C ^a  

^a YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATE; CURCUMIN; CYCLIC AMP DEPENDENT PROTEIN KINASE; GLIBENCLAMIDE; POTASSIUM CHANNEL; TRANSMEMBRANE CONDUCTANCE REGULATOR; WATER;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; APICAL MEMBRANE; ARTICLE; CYSTIC FIBROSIS; DIURESIS; ENZYME ACTIVITY; ENZYME REGULATION; FEMALE; HOMEOSTASIS; HOMOZYGOTE; KIDNEY; MOUSE; MUTATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION;

ADENOSINE TRIPHOSPHATE; ANIMALS; CURCUMIN; CYCLIC AMP-DEPENDENT PROTEIN KINASES; CYSTIC FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR; HYDROGEN-ION CONCENTRATION; KIDNEY; MICE; MICE, INBRED C57BL; MICE, INBRED CFTR; MICE, TRANSGENIC; MUTATION; OOCYTES; PATCH-CLAMP TECHNIQUES; POTASSIUM CHANNELS, INWARDLY RECTIFYING; XENOPUS LAEVIS;

EID: 33644659475     PISSN: 00219738     EISSN: 15588238     Source Type: Journal    
DOI: 10.1172/JCI26961     Document Type: Article

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