Increased expression of cFLIPL in colonic adenocarcinoma

Journal of Pathology

Volumn 194, Issue 1, 2001, Pages 15-19

  Ryu, Byung Kyu ^a   Lee, Min Goo ^a   Chi, Sung Gil ^a   Kim, Youn Wha ^a   Park, Jae Hoon ^a  

^a Kyung Hee University Hospital   (South Korea)

Author keywords

Apoptosis; cFLIP; Colon carcinoma; Immunohistochemistry; Quantitative RT PCR

Indexed keywords

APOPTOSIS INDUCING FACTOR; FAS ANTIGEN; FAS LIGAND; FLICE INHIBITORY PROTEIN; FLICE LIKE INHIBITORY PROTEIN; RNA; UNCLASSIFIED DRUG;

APOPTOSIS; ARTICLE; CANCER CELL; CANCER GROWTH; CARCINOMA CELL; CELL TRANSFORMATION; COLON ADENOCARCINOMA; COLON POLYPOSIS; CONTROLLED STUDY; DISEASE ASSOCIATION; GENE CONTROL; HUMAN; HUMAN CELL; HUMAN TISSUE; IMMUNOHISTOCHEMISTRY; IN VIVO STUDY; INTERNALIZATION; MAJOR CLINICAL STUDY; PREVALENCE; PRIORITY JOURNAL; PROTEIN EXPRESSION; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SIGNAL TRANSDUCTION; TISSUE; TUMOR GROWTH;

ADENOCARCINOMA; ADENOMATOUS POLYPS; APOPTOSIS; CARRIER PROTEINS; CASP8 AND FADD-LIKE APOPTOSIS REGULATING PROTEIN; COLONIC NEOPLASMS; GENE EXPRESSION; HUMANS; IMMUNOENZYME TECHNIQUES; INTESTINAL MUCOSA; INTRACELLULAR SIGNALING PEPTIDES AND PROTEINS; NEOPLASM PROTEINS; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION; RNA, MESSENGER; RNA, NEOPLASM;

EID: 33144488766     PISSN: 00223417     EISSN: None     Source Type: Journal    
DOI: 10.1002/path.835     Document Type: Article

References (24)

1. The molecular biology of apoptosis
2. Apoptosis in the pathogenesis and treatment of disease
3. Induction of apoptosis by monoclonal antibody anti-APO-1 class switch variants is dependent on cross-linking of APO-1 cell surface antigens
4. Molecular cloning and expression of the fas ligand, a novel member of the tumor necrosis factor family
5. Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)
6. Constitutive and induced expression of APO-1, a new member of the nerve growth factor/tumor necrosis factor receptor superfamily, in normal and neoplastic cells
7. Expression of APO-1 (CD95), a member of the NGF/TNF receptor superfamily, in normal and neoplastic colon epithelium
8. Colon carcinoma cells use different mechanisms to escape CD95-mediated apoptosis
9. The fas counterattack: Fas-mediated T cell killing by colon cancer cells expressing fas ligand
10. Anti-Fas on nonhematopoietic tumors: Levels of Fas/Apo-1 and bcl-2 are not predictive of biological resposiveness
11. Caspases: Killer proteases
12. Inhibition of death receptor signaling by FLICE-inhibitory protein as a mechanism for immune escape of tumors
13. The inhibitor of death receptor signaling, FLICE-inhibitory protein, defines a new class of tumor progression factors
14. Immune escape of tumors in vivo by expression of cellular FLICE-inhibitory protein
15. Single-step method of RNA isolation by guanidium thiocyanate-chloroform extraction
16. Alterations of the retinoblastoma gene in human prostate adenocarcinoma
17. Fas involvement in Ca(2+)- independent T cell-mediated cytotoxicity
18. Inhibition of death receptor signals by cellular FLIP
19. Modulation of caspase-8 and FLICE-inhibitory protein expression as a potential mechanism of Epstein Barr virus tumorigenesis in Burkitt's lymphoma
20. Expression of K13/v-FLIP gene of human herpesvirus 8 and apoptosis in Kaposi's sarcoma spindle cells
21. The role of c-FLIP in modulation of CD95-induced apoptosis
22. CASH, a novel caspase homologue with death effector domains
23. FLAME-1, a novel FADD-like anti-apoptotic molecule that regulates Fas/TNFRI-induced apoptosis
24. I-FLICE, a novel inhibitor of tumor necrosis factor receptor-1- and CD-95- induced apoptosis