Mast cells: Must they always be different?

Blood

Volumn 107, Issue 1, 2006, Pages 1-2

  Nechushtan, Hovav ^a   Razin, Ehud ^a  

^a HADASSAH HEBREW UNIVERSITY MEDICAL CENTER   (Israel)

Author keywords

[No Author keywords available]

Indexed keywords

CD4 ANTIGEN; COMPLEMENT COMPONENT C1Q; INTERLEUKIN 16; TETRASPANIN; VERY LATE ACTIVATION ANTIGEN 2;

ANTIGEN EXPRESSION; ASTHMA; CYTOKINE PRODUCTION; DISEASE ASSOCIATION; ENTERITIS; GRAM POSITIVE BACTERIUM; LIGAND BINDING; MAST CELL DEGRANULATION; MOLECULAR INTERACTION; NONHUMAN; PRIORITY JOURNAL; RECEPTOR AFFINITY; REVIEW; T LYMPHOCYTE ACTIVATION;

EID: 30144442234     PISSN: 00064971     EISSN: 00064971     Source Type: Journal    
DOI: 10.1182/blood-2005-10-3990     Document Type: Review

References (7)

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3. He SH. Key role of mast cells and their major secretory products in inflammatory bowel disease. World J Gastroenterol. 2004;10:309-318.
4. Williams CM, Galli SJ. Mast cells can amplify airway reactivity and features of chronic inflammation in an asthma model in mice. J Exp Med. 2000;192:455-462.
5. Qi JC, Stevens RL, Wadley R, et al. IL-16 regulation of human mast cells/basophils and their susceptibility to HIV-1. J Immunol. 2002;168:4127-4134.
6. Boucheix C, Rubinstein E. Tetraspanins. Cell Mol Life Sci. 2001;58:1189-1205.
7. Edelson BT, Li Z, Pappan LK, Zutter MM. Mast cellmediated inflammatory responses require the alpha 2 beta 1 integrin. Blood. 2004;103:2214-2220.