-
1
-
-
0037133660
-
New concepts in diastolic dysfunction and diastolic heart failure: Part I: Diagnosis, prognosis, and measurements of diastolic function
-
M.R. Zile and D.L. Brutsaert New concepts in diastolic dysfunction and diastolic heart failure: Part I: Diagnosis, prognosis, and measurements of diastolic function Circulation 105 2002 1387-1393
-
(2002)
Circulation
, vol.105
, pp. 1387-1393
-
-
Zile, M.R.1
Brutsaert, D.L.2
-
2
-
-
0034595408
-
Defining diastolic dysfunction
-
W. Grossman Defining diastolic dysfunction Circulation 101 2000 2020-2021
-
(2000)
Circulation
, vol.101
, pp. 2020-2021
-
-
Grossman, W.1
-
3
-
-
1642542466
-
Hypertensive myocardial fibrosis and diastolic dysfunction: Another model of inflammation?
-
F. Kuwahara et al. Hypertensive myocardial fibrosis and diastolic dysfunction: Another model of inflammation? Hypertension 43 2004 739-745
-
(2004)
Hypertension
, vol.43
, pp. 739-745
-
-
Kuwahara, F.1
-
4
-
-
0038460302
-
The Seventh Report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure: The JNC 7 Report
-
A.V. Chobanian et al. The Seventh Report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure: The JNC 7 Report JAMA 289 2003 2560-2571
-
(2003)
JAMA
, vol.289
, pp. 2560-2571
-
-
Chobanian, A.V.1
-
5
-
-
2642587418
-
Diastolic heart failure: Challenges of diagnosis and treatment
-
C. Gutierrez and D.G. Blanchard Diastolic heart failure: Challenges of diagnosis and treatment Am. Fam. Physician 69 2004 2609-2616
-
(2004)
Am. Fam. Physician
, vol.69
, pp. 2609-2616
-
-
Gutierrez, C.1
Blanchard, D.G.2
-
6
-
-
3543099842
-
Effect of aldosterone antagonism on myocardial dysfunction in hypertensive patients with diastolic heart failure
-
P.M. Mottram et al. Effect of aldosterone antagonism on myocardial dysfunction in hypertensive patients with diastolic heart failure Circulation 110 2004 558-565
-
(2004)
Circulation
, vol.110
, pp. 558-565
-
-
Mottram, P.M.1
-
7
-
-
0036272927
-
Myocardial stiffness is determined by ventricular fibrosis, but not by compensatory or excessive hypertrophy in hypertensive heart
-
K. Yamamoto et al. Myocardial stiffness is determined by ventricular fibrosis, but not by compensatory or excessive hypertrophy in hypertensive heart Cardiovasc. Res. 55 2002 76-82
-
(2002)
Cardiovasc. Res.
, vol.55
, pp. 76-82
-
-
Yamamoto, K.1
-
8
-
-
0346727114
-
Effects of ACE inhibition versus non-ACE inhibitor antihypertensive treatment on myocardial fibrosis in patients with arterial hypertension
-
C. Brilla et al. Effects of ACE inhibition versus non-ACE inhibitor antihypertensive treatment on myocardial fibrosis in patients with arterial hypertension Herz 28 2003 744-753
-
(2003)
Herz
, vol.28
, pp. 744-753
-
-
Brilla, C.1
-
9
-
-
13844312743
-
The cardiac fibroblast: Therapeutic target in myocardial remodeling and failure
-
R.D. Brown et al. The cardiac fibroblast: Therapeutic target in myocardial remodeling and failure Annu. Rev. Pharmacol. Toxicol. 45 2005 657-687
-
(2005)
Annu. Rev. Pharmacol. Toxicol.
, vol.45
, pp. 657-687
-
-
Brown, R.D.1
-
10
-
-
0034727695
-
Limitation of excessive extracellular matrix turnover contribute to survival benefit of spironolactone therapy in patients with congestive heart failure: Insights from the randomized aldactone evaluation study (RALES)
-
RALES Investigators
-
F. Zannad et al. Limitation of excessive extracellular matrix turnover contribute to survival benefit of spironolactone therapy in patients with congestive heart failure: Insights from the randomized aldactone evaluation study (RALES). RALES Investigators Circulation 102 2000 2700-2706
-
(2000)
Circulation
, vol.102
, pp. 2700-2706
-
-
Zannad, F.1
-
11
-
-
0033745462
-
CTGF expression is induced by TGF-beta in cardiac fibroblasts and cardiac myocytes: A potential role in heart fibrosis
-
M.M. Chen et al. CTGF expression is induced by TGF-beta in cardiac fibroblasts and cardiac myocytes: A potential role in heart fibrosis J. Mol. Cell. Cardiol. 32 2000 1805-1819
-
(2000)
J. Mol. Cell. Cardiol.
, vol.32
, pp. 1805-1819
-
-
Chen, M.M.1
-
12
-
-
1542358803
-
Connective tissue growth factor - A novel mediator of angiotensin II-stimulated cardiac fibroblast activation in heart failure in rats
-
M.S. Ahmed et al. Connective tissue growth factor - a novel mediator of angiotensin II-stimulated cardiac fibroblast activation in heart failure in rats J. Mol. Cell. Cardiol. 36 2004 393-404
-
(2004)
J. Mol. Cell. Cardiol.
, vol.36
, pp. 393-404
-
-
Ahmed, M.S.1
-
13
-
-
4844224644
-
Gene expression changes associated with fibronectin-induced cardiac myocyte hypertrophy
-
H. Chen et al. Gene expression changes associated with fibronectin-induced cardiac myocyte hypertrophy Physiol. Genomics 18 2004 273-283
-
(2004)
Physiol. Genomics
, vol.18
, pp. 273-283
-
-
Chen, H.1
-
14
-
-
18444384225
-
Induction of myocardial connective tissue growth factor in pacing-induced heart failure in pigs
-
M.S. Ahmed et al. Induction of myocardial connective tissue growth factor in pacing-induced heart failure in pigs Acta Physiol. Scand. 184 2005 27-36
-
(2005)
Acta Physiol. Scand.
, vol.184
, pp. 27-36
-
-
Ahmed, M.S.1
-
15
-
-
20944432483
-
Differential regulation of angiotensin II-induced expression of connective tissue growth factor by protein kinase C isoforms in the myocardium
-
Z. He et al. Differential regulation of angiotensin II-induced expression of connective tissue growth factor by protein kinase C isoforms in the myocardium J. Biol. Chem. 280 2005 15719-15726
-
(2005)
J. Biol. Chem.
, vol.280
, pp. 15719-15726
-
-
He, Z.1
-
16
-
-
0036645337
-
Transforming growth factor-{beta} function blocking prevents myocardial fibrosis and diastolic dysfunction in pressure-overloaded rats
-
F. Kuwahara et al. Transforming growth factor-{beta} function blocking prevents myocardial fibrosis and diastolic dysfunction in pressure-overloaded rats Circulation 106 2002 130-135
-
(2002)
Circulation
, vol.106
, pp. 130-135
-
-
Kuwahara, F.1
-
17
-
-
0036198371
-
Tumor necrosis factor-alpha and myocardial remodeling in progression of heart failure: A current perspective
-
W.S. Bradham et al. Tumor necrosis factor-alpha and myocardial remodeling in progression of heart failure: A current perspective Cardiovasc. Res. 53 2002 822-830
-
(2002)
Cardiovasc. Res.
, vol.53
, pp. 822-830
-
-
Bradham, W.S.1
-
18
-
-
0031778559
-
The role of the neurohormonal system in heart failure
-
M. Komajda et al. The role of the neurohormonal system in heart failure Heart 79 1998 17S-23S
-
(1998)
Heart
, vol.79
-
-
Komajda, M.1
-
19
-
-
0034307880
-
Endothelin-receptor antagonists: Current and future perspectives
-
A. Ray et al. Endothelin-receptor antagonists: Current and future perspectives Drug Discov. Today 5 2000 455-464
-
(2000)
Drug Discov. Today
, vol.5
, pp. 455-464
-
-
Ray, A.1
-
20
-
-
4444315888
-
Matricellular proteins in the heart: Possible role during stress and remodeling
-
M.W.M. Schellings et al. Matricellular proteins in the heart: Possible role during stress and remodeling Cardiovasc. Res. 64 2004 24-31
-
(2004)
Cardiovasc. Res.
, vol.64
, pp. 24-31
-
-
Schellings, M.W.M.1
-
21
-
-
4444281166
-
Cell-matrix signaling and thrombospondin: Another link to myocardial matrix remodeling
-
F.G. Spinale Cell-matrix signaling and thrombospondin: Another link to myocardial matrix remodeling Circ. Res. 95 2004 446-448
-
(2004)
Circ. Res.
, vol.95
, pp. 446-448
-
-
Spinale, F.G.1
-
22
-
-
4444312145
-
Thrombospondin-2 is essential for myocardial matrix integrity: Increased expression identifies failure-prone cardiac hypertrophy
-
B. Schroen et al. Thrombospondin-2 is essential for myocardial matrix integrity: Increased expression identifies failure-prone cardiac hypertrophy Circ. Res. 95 2004 515-522
-
(2004)
Circ. Res.
, vol.95
, pp. 515-522
-
-
Schroen, B.1
-
23
-
-
0036728623
-
MMPs and TIMPs - An historical perspective
-
J.F. Woessner Jr. MMPs and TIMPs - an historical perspective Mol. Biotechnol. 22 2002 33-49
-
(2002)
Mol. Biotechnol.
, vol.22
, pp. 33-49
-
-
Woessner Jr., J.F.1
-
24
-
-
0036084756
-
MMP inhibition modulates TNF-alpha transgenic mouse phenotype early in the development of heart failure
-
Y.Y. Li et al. MMP inhibition modulates TNF-alpha transgenic mouse phenotype early in the development of heart failure Am. J. Physiol. Heart Circ. Physiol. 282 2002 H983-H989
-
(2002)
Am. J. Physiol. Heart Circ. Physiol.
, vol.282
-
-
Li, Y.Y.1
-
25
-
-
0036322309
-
TIMP-1: A marker of left ventricular diastolic dysfunction and fibrosis in hypertension
-
M.M. Lindsay et al. TIMP-1: A marker of left ventricular diastolic dysfunction and fibrosis in hypertension Hypertension 40 2002 136-141
-
(2002)
Hypertension
, vol.40
, pp. 136-141
-
-
Lindsay, M.M.1
-
26
-
-
13244271256
-
Tissue inhibitor of metalloproteinse-1 is a marker of diastolic dysfunction using tissue Doppler in patients with type 2 diabetes and hypertension
-
M.H. Tayebjee et al. Tissue inhibitor of metalloproteinse-1 is a marker of diastolic dysfunction using tissue Doppler in patients with type 2 diabetes and hypertension Eur. J. Clin. Invest. 35 2005 8-12
-
(2005)
Eur. J. Clin. Invest.
, vol.35
, pp. 8-12
-
-
Tayebjee, M.H.1
-
27
-
-
16244371321
-
The effect of alagebrium chloride (ALT-711), a novel glucose cross-link breaker, in the treatment of elderly patients with diastolic heart failure
-
W.C. Little et al. The effect of alagebrium chloride (ALT-711), a novel glucose cross-link breaker, in the treatment of elderly patients with diastolic heart failure J. Card. Fail. 11 2005 191-195
-
(2005)
J. Card. Fail.
, vol.11
, pp. 191-195
-
-
Little, W.C.1
-
28
-
-
0345303768
-
Glycation end-product cross-link breaker reduces collagen and improves cardiac function in aging diabetic heart
-
J. Liu et al. Glycation end-product cross-link breaker reduces collagen and improves cardiac function in aging diabetic heart Am. J. Physiol. Heart Circ. Physiol. 285 2003 H2587-H2591
-
(2003)
Am. J. Physiol. Heart Circ. Physiol.
, vol.285
-
-
Liu, J.1
-
29
-
-
7044231909
-
Attenuation of extracellular matrix accumulation in diabetic nephropathy by the advanced glycation end product cross-link breaker ALT-711 via a protein kinase C-{alpha}-dependent pathway
-
V. Thallas-Bonke et al. Attenuation of extracellular matrix accumulation in diabetic nephropathy by the advanced glycation end product cross-link breaker ALT-711 via a protein kinase C-{alpha}-dependent pathway Diabetes 53 2004 2921-2930
-
(2004)
Diabetes
, vol.53
, pp. 2921-2930
-
-
Thallas-Bonke, V.1
-
30
-
-
0142121508
-
Therapeutic potential of breakers of advanced glycation end product-protein crosslinks
-
S. Vasan et al. Therapeutic potential of breakers of advanced glycation end product-protein crosslinks Arch. Biochem. Biophys. 419 2003 89-96
-
(2003)
Arch. Biochem. Biophys.
, vol.419
, pp. 89-96
-
-
Vasan, S.1
-
31
-
-
0035949502
-
Improved arterial compliance by a novel advanced glycation end-product crosslink breaker
-
D.A. Kass et al. Improved arterial compliance by a novel advanced glycation end-product crosslink breaker Circulation 104 2001 1464-1470
-
(2001)
Circulation
, vol.104
, pp. 1464-1470
-
-
Kass, D.A.1
-
32
-
-
0041315638
-
The receptor RAGE as a progression factor amplifying arachidonate-dependent inflammatory and proteolytic response in human atherosclerotic plaques: Role of glycemic control
-
F. Cipollone et al. The receptor RAGE as a progression factor amplifying arachidonate-dependent inflammatory and proteolytic response in human atherosclerotic plaques: Role of glycemic control Circulation 108 2003 1070-1077
-
(2003)
Circulation
, vol.108
, pp. 1070-1077
-
-
Cipollone, F.1
|