Endothel function and ischemic heart disease;Endothelfunkció és ischaemiás szí vbetegség

Orvosi Hetilap

Volumn 146, Issue 20 SUPPL. 2, 2005, Pages 1047-1053

  Préda, István ^a,b   Bencze, Jusztina ^a   Vargova, Katarina ^a  

^a SEMMELWEIS UNIVERSITY   (Hungary)

^b NONE   (Hungary)

Author keywords

Atherosclerosis; Atherothrombosis; Endothelial dysfunction; Laboratory methods for endothelial functions; Plaque rupture

Indexed keywords

LOW DENSITY LIPOPROTEIN; CELL ADHESION MOLECULE; CYTOKINE; HOMOCYSTEINE;

ANGIOCARDIOGRAPHY; ARTICLE; ATHEROSCLEROTIC CARDIOVASCULAR DISEASE; ATHEROSCLEROTIC PLAQUE; CARDIOVASCULAR RISK; CELL ADHESION; CELL FUNCTION; DISEASE COURSE; ENDOTHELIAL DYSFUNCTION; ENDOTHELIUM CELL; HUMAN; INFLAMMATION; ISCHEMIC HEART DISEASE; LASER DOPPLER FLOWMETRY; OXIDATION; PATHOGENESIS; VASODILATATION; ARTERITIS; BLOOD; BRACHIAL ARTERY; ECHOGRAPHY; HEART MUSCLE ISCHEMIA; HYPERCHOLESTEROLEMIA; HYPERHOMOCYSTEINEMIA; HYPERTENSION; IMMUNOLOGY; INFECTION; METABOLISM; MONOCYTE; OXIDATION REDUCTION REACTION; PATHOPHYSIOLOGY; REVIEW; RISK FACTOR; THROMBOCYTE ADHESIVENESS; THROMBOCYTE AGGREGATION; VASCULAR ENDOTHELIUM;

ARTERITIS; BRACHIAL ARTERY; CELL ADHESION MOLECULES; CORONARY ANGIOGRAPHY; CYTOKINES; ENDOTHELIUM, VASCULAR; HOMOCYSTEINE; HUMANS; HYPERCHOLESTEROLEMIA; HYPERHOMOCYSTEINEMIA; HYPERTENSION; INFECTION; INFLAMMATION; LASER-DOPPLER FLOWMETRY; LIPOPROTEINS, LDL; MONOCYTES; MYOCARDIAL ISCHEMIA; OXIDATION-REDUCTION; PLATELET ADHESIVENESS; PLATELET AGGREGATION; RISK FACTORS; VASODILATION;

EID: 22144471213     PISSN: 00306002     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (55)

1. Bencze, J., Hízoh, I., Kiss, R. G. és mtsa: Az endothelfunkció zavara szívbetegekben. Klasszifikácio, diagnosztikus és terápiás lehetoségek. Orvosképzés, 2001, 76, 210-220.
2. Bencze, J., Kiss, R. G., Korda, A. és mtsai: Angiographic and biochemical findings in patients with "slow" coronary flow and myocardial ischemia. Eur. Heart J. 2004, (Suppl.) 343. (Abst.)
3. Bencze, J., Kiss, R. G., Tóth-Zsámboki, E. és mtsai: Coronary slow flow phenomenon as a subtype of ischemic heart disease can be characterized by clinical and angiographic findings, endothelial and inflammatory markers (közlés alatt).
4. Bombeli, T., Schwarcz, T. T., Harlan, J. M.: Adhesion of activated platelets to endothelial cells: evidence for a GPIIbIIIa dependent bridging mechanism and novel roles for endothelial intercellular adhesion molecule-1 (ICAM-1), alfavbeta3 integrin and GP Ib (alpha). J. Exp. Med., 1998, 187, 329-339.
5. Cannon, C. P., McCabe, C. H., Beider, R. és mtsai: Design of the Pravastatin or Atorvastatin Evaluation and infection (PROVE IT)-TIMI 22 trial. Am. J. Cardiol., 2002, 89, 860-866.
6. Celermajer, D. S., Sorensen, K. E., Bull, C. és mtsai: Endothelium dependent dilation in the systemic arteries of asymptomatic subjects relates to coronarz risk factors and their interaction. J. Am. Coll. Card., 1994, 24, 1468-1474.
7. Chang, M. Y., Sasahara, M., Chait, A: Inhibition of hyperchpelsterolaemia induced atherosclerosis in nonhumat primate by probucol. Atheroscler. Thromb. Vasc. Biol., 1995, 15, 1631-1640.
8. Chobanian.A. V., Dzau, V. J.: Renin angiotensin system and atherosclerotic vascular disease. In Atherosclerosis and Coronary Heart Disease. Vol. 1. Szerk.: Fuster, V., Rorr, E., Topol, R. J. Philadelphia, Lippincott-Raven, 1996, 237-242 old.
9. Cines, D. B., Pollack, E. S., Buck, A. és mtsai: Endothelial cells in physiology and pathophysyology of vascular disorders. Blood, 1998, 91, 3527-3561.
10. Davies, M. J.: A macro and micro view of coronary vascular insult in ischemic heart disease. Circulation, 1990, 82 (Suppl.) II-38-II-48.
11. Falk, E., Shah, P. K., Fuster, V.: Pathogenesis of plaque disruption. In Atherosclerosis and Coronary Artery Disease. Vol 2. Szerk.: Fuster, V., Ross, R., Topol, E. J. Lippincott-Raven Philadelphia, 1996, 492-510. old.
12. Farkas, K.: Muszeres vizsgálatok érbetegségekben In Vascularis Medicina. Szerk.: Kleskó, E. Therápia Kiadó, 2004, 128-147.
13. Farkas, K., Kolossváry, Z., Járai, Z. és mtsai: Non-invasive assessment of microvascular endothel function by laser Doppler flowmetry in patients with essential hypertension. Atherosclerosis, 2004, 173, 97-102.
14. Folcik, V. A., Aamir, R., Cathcart, M. K.: Cytokine modulation of LDL oxydation by activated humen mondcytes. Atheroscler. Thromb. Vasc. Biol. 1997, 17, 1954-1961.
15. George, F., Brisson, C., Poncelet, P. és mtsai: Rapid isolation of human endothelial cells from whole blood using S-endo 1 monoclonal antibody coupled to immuno-magnetic beads: Demonstration of endothelial injury after coronary angioplasty. Thromb. Haemost. 1992, 76, 147-153.
16. Grayston, J. T.: Secondary prevention antibiotic treatment trials for coronary artery disease. Circulation, 2003, 102, 1742-1750.
17. Gibbons, G. H., Pratt, R. E., Dzau, V. J.: Vascular smooth muscle cell hypertrophia vs hyperplasia: autocrin transforming growth factor-beta-1 expression determines growth response of angiotensin II. J. Clin. Invest., 1992, 90, 456-461.
18. Glagov, S., Weisenberg, E., Zarins, C. K. és mtsa: Compensatory enlargement of human atherosclerotic arteries. N. Engl. J. Med., 1987, 316, 1371-1375.
19. Griendling, K. K., Alexander, R. W.: Oxydative stress and cardiovascular disease. Circulation, 1997, 96, 3264-3265.
20. Hajjar, K. A.: Homocysteine-induced modulation of tissue plasminogen activator binding to its endothelial cell membrane receptor. J. Clin. Invest., 1993, 91, 2873-2879.
21. Hajjar, D. P., Haberland, M. E.: Lipoprotein trafficking in vascular cells: molecular Trojan horses and cellular saboteurs. J. Biol. Chem., 1997, 272, 22975-22978.
22. Hladovecz, J.: Circulating endothelial cells as a sign of vessel wall lesion. Physiol. Bohemoslov, 1978, 27, 140-144.
23. Harker, L. A., Ross, R., Slichter, S. J. és mtsa: Homocysteine-induced arteriosclerosis: the role of endothelial cell injury and platelet response in its genesis. J. Clin. Invest., 1976, 58, 731-741.
24. Hollenbaugh, D., Mischel-Petty, N., Edwards, C. P. és mtsai: Expression of functional CD40 by vascular endothelial cells. J. Exp. Med., 1995, 182, 33-40.
25. Kubli, S., Waeber, B., Dalla-Eve, A. és mtsa: Reproducibility of laser Doppler imaging of skin blood flow as a tool to assess endothelial function. J. Cardiovasc. Pharmacol., 2000, 36, 640-648.
26. Kalayoglu, M. V., Byrne, G. I. Induction of macrophage foam cell formation by Chlamydia pneumoniae. J. Infect. Dis., 1998, 177, 725-731.
27. Johansson, L., Holm, J., Skalli, O. és mtsai: Regional accumulation of T cells, macrophages, and smooth muscle cells in the human atherosclerotic plaque. Atherosclerosis, 1986, 6, 131-138.
28. Libby, P., Ross, R.: Cytokines and growth regulatory molecules. In Atherosclerosis and Coronary Artery Disease. Vol 1. Szerk.: Fuster, V., Ross, R., Topol, E. J. Lippincott-Raven, Philadelphia, 1996, 585-594. old.
29. Libby, P., Egan, D., Skarlatos, R.: Roles of infectious agents in atherosclerosis and restenosis: An assessment of evidences and need for future research. Circulation, 1997, 96, 495-501.
30. Lüscher, T., Barton, M.: Biology of the Endothelium. Clin. Cardiol., 1997, 20, 3-10.
31. Majors, A., Ehrhart, L. A., Pazecka, E. H.: Homocysteine as a risk factor for vascular disease: enhanced collagen production and accumulation by smooth muscle cells. Atheroscler. Thromb. Vasc. Biol., 1997, 17, 2074-2081.
32. McCully, K. S.: Vascular pathology of homocysteinaemia: implications for the pathogenesis of arteriosclerosis. Am. J. Pathol., 1969, 56, 111-128.
33. Moss, M. L., Jin, S. L.-C., Milla, M. E. és mtsai: Cloning of disintegrin metalloproteinase that processes precursor tumor necrosis factor-(alpha). Nature, 1997, 385, 733-736.
34. Nagy, L., Bajkó, S., Fábián, E. és mtsai: Magyar konszenzus az aneria brachiális áramlá sfüggo "flow mediated" vasodilataciójának vizsgálatához. Érbetegségek, 2003, 10, 47-50.
35. Nieszner, É., Bárdos, B., Baranyi, É. és mtsa: A diabetes mellitus kardiovaszkuláris szovodményei és kezelése. Lege Artis Medicinae (közlés alatt)
36. O'Connor, S. S., Taylor, C., Campbell, L. A. és mtsai: Potential infectious etiologies of atherosclerosis: a multifactorial perspective. Emerg. Infect. Dis., 2001, 7, 780-788.
37. O'Connor, C. M., Dunne, M. V., Pfeffer, M. és mtsai: Azithromycin for the secondary prevention of coronary heart disease events: Ther WISARD study: A randomized controlled trial. JAMA, 2003, 290, 1459-1466.
38. Nunes, G. L., Robinson, K., Kalynych, A. és mtsai: Vitamin C and E inhibit o2-production in the pig coronary artery. Circulation, 1997, 96, 3593-3601.
39. Raines, E. W., Rosenfeld, M. E., Ross, R.: The role of macrophages. In Atherosclerosis and coronary artery disease. Vol 1. Szerk.: Fuster, V., Ross, R., Topol, E. J. Lippincott-Raven, Philadelphia, 1996, 539-555. old.
40. Ross, R., Glomest, J. A.: Atherosclerosis and the arterial smooth muscle cell: proliferation of smooth muscle is a key event in the genesis of the lesions of atherosclerosis. Science, 1973, 180, 1332-1339.
41. Ross, R.: The pathogenesis of atherosclerosis: a perspective for the 1990s. Nature, 1993, 362, 801-809.
42. Ross, R.: Mechanism of disease: Atherosclerosis - an inflammatory disease. N. Engl. J. Med., 1999, 340, 115-126.
43. Stary, H. C., Chandler, A. B., Glagov, S. és mtsai: A definition of initial, fatty streak and intermediate lesions of atherosclerosis: a report from the Committee on Vascular Lesions of the Council of Atherosclerosis, American Heart Association. Circulation, 1994, 89, 2462-2478.
44. Stary, H. C.: The histological classification of atherosclerotic lesions in human coronary arteries. Atherosclerosis and Coronary Artery Disease. Vol 1. Szerk.: Fuster, V., Ross, R., Topol, E. J. Philadelphia, Lippincott-Raven, 1996, 463-474. old.
45. Stephens, N. G., Parsons, A., Schofield, P. M. és mtsai: Randomised controlled trial of vitamin-E in patients with coronary disease: Cambridge Heart Antioxydant Study. Lancet, 1996, 347, 781-786.
46. Springer, T. A., Cybusky, M. I.: Traffic signals on endothelium leukocytes for health, inflammation and atherosclerosis. In Atherosclerosis and coronary artery disease. Vol. 1. Szerk.: Fuster, V., Ross, R., Topol, E. J. Philadelphia, Lippincott-Raven, 1996, 596-606.
47. Stemme, S., Faber, B., Holm, J. és mtsai: T lymphocytes from human atherosclerotic plaques recognize oxidized low density lipoprotein. Proc. Nat. Acad. Sci. USA, 1995, 92, 3893-3897.
48. Upchurch, G. R. Jr., Welch, G. N., Fabian, A. J. és mtsai: Homocyst(e)ine decreases bioavailable nitric oxide by a mechanism involving glutathion peroxidase. J. Biol. Chem., 1997, 272, 171012-171017.
49. Van der Wal, A. C., Das, P. K., Betz van der Berg, és mtsai: Atherosclerotic lesions in humans: in situ immunophenotyic analysis suggesting an immune mediated response. Lab. Invest., 1989, 61, 166-170.
50. Vargová, K., Tóth-Zsámboki, E., Csóti, É. és mtsai: Keringo humán endothelsejtek izolálása és immunohisztokémiai analízise atherothrombotikus állapotokban. Magyar Belorvosi Archívum, 2004, 2S: 28-39.
51. Verhoef, P., Stampfer, M. J.: Prospective studies of homcysteine and cardiovascular disease. Nutr. Rev., 1995, 53, 283-288.
52. Veres, A., Füst, Gy., Snmieja, M. és mtsai: Relationship of anti-60kDa heat shock protein and anticholesterol-antibodies to cardiovascular events. Circulation, 2002, 106, 2775-2780.
53. Voelker, W., Euchner, U., Dittmann, H. és mtsa: Long-term clinical course of patients with angina and angiographically normal coronary arteries. Clin. Cardiol., 1991, 14, 307-311.
54. Vogel, R. A.: Measurement of endothelial function by brachial artery flow-mediated vasodilation. Am. Cardiol., 2001, 88, 2A 31E-34E.
55. Wick, G., Romen, R., Amberger, A. és mtsai: Atherosclerosis, autoimmunity, and vascular associated lymphoid tissue. FASEB J., 1997, 11, 1199-1207.