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Volumn 29, Issue 1, 2001, Pages 185-196
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Disruption of CIC-3, a chloride channel expressed on synaptic vesicles, leads to a loss of the hippocampus
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Author keywords
[No Author keywords available]
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Indexed keywords
2 AMINO 5 PHOSPHONOVALERIC ACID;
6 NITRO 7 SULFAMOYLBENZO[F]QUINOXALINE 2,3 DIONE;
BICUCULLINE;
CHLORIDE CHANNEL;
CHLORIDE ION;
ION CHANNEL;
POTASSIUM CHLORIDE;
TETRODOTOXIN;
ACID;
CHLORIDE;
CLC 3 CHANNEL;
CLC-3 CHANNEL;
ANIMAL CELL;
ARTICLE;
CENTRAL NERVOUS SYSTEM;
ELECTRORETINOGRAPHY;
HIPPOCAMPUS;
NEUROTRANSMISSION;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
RETINA DEGENERATION;
SYNAPSE VESICLE;
ANIMAL;
ANIMAL BEHAVIOR;
BIOSYNTHESIS;
EXCITATORY POSTSYNAPTIC POTENTIAL;
GENE TARGETING;
GENETICS;
GROWTH DISORDER;
IN VITRO STUDY;
METABOLISM;
MOTOR ACTIVITY;
MOUSE;
MOUSE MUTANT;
PATHOLOGY;
PATHOPHYSIOLOGY;
PYRAMIDAL NERVE CELL;
ACIDS;
ANIMALS;
BEHAVIOR, ANIMAL;
CHLORIDE CHANNELS;
CHLORIDES;
ELECTRORETINOGRAPHY;
EXCITATORY POSTSYNAPTIC POTENTIALS;
GENE TARGETING;
GROWTH DISORDERS;
HIPPOCAMPUS;
MICE;
MICE, KNOCKOUT;
MOTOR ACTIVITY;
PYRAMIDAL CELLS;
RETINAL DEGENERATION;
SYNAPTIC VESICLES;
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EID: 17744375755
PISSN: 08966273
EISSN: None
Source Type: Journal
DOI: 10.1016/S0896-6273(01)00189-1 Document Type: Article |
Times cited : (447)
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References (54)
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