Aβ(31-35) peptide induce apoptosis in PC 12 cells: Contrast with Aβ(25-35) peptide and examination of underlying mechanisms

Neurochemistry International

Volumn 46, Issue 7, 2005, Pages 575-583

  Misiti, Francesco ^a   Sampaolese, Beatrice ^b   Pezzotti, Michela ^a   Marini, Stefano ^c   Coletta, Massimo ^c   Ceccarelli, Lia ^a   Giardina, Bruno ^a,b   Clementi, Maria Elisabetta ^b  

^a CATHOLIC UNIVERSITY   (Italy)

^b CNR   (Italy)

^c UNIVERSITY OF ROME TOR VERGATA   (Italy)

Author keywords

Amyloid peptide; Apoptosis; A P(25 35) fragment; A P(31 35) fragment; Methionine oxidation; Mitochondria; Neurotoxicity

Indexed keywords

3 [(4,5 DIMETHYLTHIAZOL 2 YL) 5,3 CARBOXYMETHOXYPHENYL] 2 (4 SULFOPHENYL) 2H TETRAZOLIUM; AMYLOID BETA PROTEIN; AMYLOID BETA PROTEIN[25-35]; AMYLOID BETA PROTEIN[31-35]; AMYLOID PROTEIN; CASPASE; DNA FRAGMENT; TETRAZOLIUM; UNCLASSIFIED DRUG; AMYLOID BETA PROTEIN (25 35); AMYLOID BETA PROTEIN (31 35); AMYLOID BETA-PROTEIN (25-35); AMYLOID BETA-PROTEIN (31-35); CASP3 PROTEIN, RAT; CASPASE 3; METHIONINE; PEPTIDE FRAGMENT;

AMINO ACID SEQUENCE; ANIMAL CELL; APOPTOSIS; ARTICLE; ASSAY; CELL DEATH; CELL STRAIN; CONTROLLED STUDY; CYTOTOXICITY; ENZYME ACTIVATION; EVALUATION; NEUROTOXICITY; NONHUMAN; PEPTIDE ANALYSIS; PRIORITY JOURNAL; RAT; SEQUENCE ANALYSIS; ALZHEIMER DISEASE; ANIMAL; BRAIN; CELL SURVIVAL; CHEMICALLY INDUCED DISORDER; COMPARATIVE STUDY; DRUG EFFECT; METABOLISM; NERVE CELL; NERVE DEGENERATION; OXIDATION REDUCTION REACTION; OXIDATIVE STRESS; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; ANIMALS; APOPTOSIS; BRAIN; CASPASE 3; CASPASES; CELL SURVIVAL; DNA FRAGMENTATION; METHIONINE; NERVE DEGENERATION; NEURONS; OXIDATION-REDUCTION; OXIDATIVE STRESS; PC12 CELLS; PEPTIDE FRAGMENTS; RATS;

EID: 17544372317     PISSN: 01970186     EISSN: None     Source Type: Journal    
DOI: 10.1016/j.neuint.2005.01.001     Document Type: Article

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