-
1
-
-
0343809698
-
Ca channels in the cardiovascular system
-
Futura Publishing, Mount Kisco, NY
-
R.W. Tsien, M.R. Rosen, M.J. Janse, A.L. Wit, Ca channels in the cardiovascular system, in: Cardiac Electrophysiology: A Textbook, Futura Publishing, Mount Kisco, NY, 1990, pp. 75-89.
-
(1990)
Cardiac Electrophysiology: A Textbook
, pp. 75-89
-
-
Tsien, R.W.1
Rosen, M.R.2
Janse, M.J.3
Wit, A.L.4
-
2
-
-
0028297589
-
Regulation and modulation of Ca channels in cardiac, skeletal, and smooth muscle cells
-
McDonald T.F., Pelzer S., Trautwein W., Pelzer D.J. Regulation and modulation of Ca channels in cardiac, skeletal, and smooth muscle cells. Physiol. Rev. 74:1994;365-507.
-
(1994)
Physiol. Rev.
, vol.74
, pp. 365-507
-
-
McDonald, T.F.1
Pelzer, S.2
Trautwein, W.3
Pelzer, D.J.4
-
4
-
-
0003559343
-
-
Orlando, FL: Academic Press Inc. 179-205
-
Leach K.L., Blumberg P.M., Michael R.H., Drummond A.H., Downs C.P. Inositol Lipids in Cell Signalling. 1989;Academic Press Inc, Orlando, FL. 179-205.
-
(1989)
Inositol Lipids in Cell Signalling
-
-
Leach, K.L.1
Blumberg, P.M.2
Michael, R.H.3
Drummond, A.H.4
Downs, C.P.5
-
6
-
-
0030055616
-
1 -Adrenergic activation inhibits β-adrenergic stimulated unitary Ca currents in cardiac ventricular myocytes
-
1 -Adrenergic activation inhibits β-adrenergic stimulated unitary Ca currents in cardiac ventricular myocytes Circ. Res. 79:1996;184-193.
-
(1996)
Circ. Res.
, vol.79
, pp. 184-193
-
-
Chen, L.1
El Sherif, N.2
Boutjdir, M.3
-
8
-
-
0034533553
-
Evidence for functional role of epsilon PKC isozyme in the regulation of cardiac Ca channels
-
Hu K., Mochly-Rosen D., Boutjdir M. Evidence for functional role of epsilon PKC isozyme in the regulation of cardiac Ca channels. Am. J. Physiol. Heart Circ. Physiol. 279:2000;H2658-H2664.
-
(2000)
Am. J. Physiol. Heart Circ. Physiol.
, vol.279
-
-
Hu, K.1
Mochly-Rosen, D.2
Boutjdir, M.3
-
9
-
-
0032917447
-
Increased protein kinase C activity and expression of Ca sensitive isoforms in the failing human heart
-
Bowling N., Walsh R.A., Song G., Estridge T., Sandusky G.E., Fouts R.L., Mintze K., Pickard T., Roden R., Bristow M.R., Sabbah N.H., Mizrahi J.L., Gromo G., King G.L., Vlahos C.J. Increased protein kinase C activity and expression of Ca sensitive isoforms in the failing human heart. Circulation. 99:1999;384-391.
-
(1999)
Circulation
, vol.99
, pp. 384-391
-
-
Bowling, N.1
Walsh, R.A.2
Song, G.3
Estridge, T.4
Sandusky, G.E.5
Fouts, R.L.6
Mintze, K.7
Pickard, T.8
Roden, R.9
Bristow, M.R.10
Sabbah, N.H.11
Mizrahi, J.L.12
Gromo, G.13
King, G.L.14
Vlahos, C.J.15
-
10
-
-
0030055615
-
Protein kinase C isoform expression in normal and failing rabbit hearts
-
Rout-Benzineb P., Mohammadi K., Perennec J., Payard M., Bouanani N.E.H., Crozatier B. Protein kinase C isoform expression in normal and failing rabbit hearts. Circ. Res. 79:1996;153-161.
-
(1996)
Circ. Res.
, vol.79
, pp. 153-161
-
-
Rout-Benzineb, P.1
Mohammadi, K.2
Perennec, J.3
Payard, M.4
Bouanani, N.E.H.5
Crozatier, B.6
-
11
-
-
0032901397
-
Effect of angiotensin-converting enzyme inhibition on protein kinase C and SR proteins in heart failure
-
Takeishi Y., Bhagwat A., Ball N.A., Kirkpatrick D.L., Periyasami M., Walsh R.A. Effect of angiotensin-converting enzyme inhibition on protein kinase C and SR proteins in heart failure. Am. J. Physiol. Heart Circ. Physiol. 276:1999;H53-H62.
-
(1999)
Am. J. Physiol. Heart Circ. Physiol.
, vol.276
-
-
Takeishi, Y.1
Bhagwat, A.2
Ball, N.A.3
Kirkpatrick, D.L.4
Periyasami, M.5
Walsh, R.A.6
-
12
-
-
0032802635
-
Alterations in protein kinase a and protein kinase C levels in heart failure due to genetic cardiomyopathy
-
Wang J., Liu X., Arneja A.S., Dhalla N.S. Alterations in protein kinase A and protein kinase C levels in heart failure due to genetic cardiomyopathy. Can. J. Cardiol. 15:1999;683-690.
-
(1999)
Can. J. Cardiol.
, vol.15
, pp. 683-690
-
-
Wang, J.1
Liu, X.2
Arneja, A.S.3
Dhalla, N.S.4
-
13
-
-
0034625662
-
Cardiotrophic effects of PKC epsilon: Analysis by in vivo modulation of PKC epsilon translocation
-
Mochly-Rosen D.G., Wu H., Hahn H., Osinska T., Liron J.N., Lorenz A., Yatani J., Robbins G.W., Dorn II Cardiotrophic effects of PKC epsilon: analysis by in vivo modulation of PKC epsilon translocation. Circ. Res. 86:2000;1173-1179.
-
(2000)
Circ. Res.
, vol.86
, pp. 1173-1179
-
-
Mochly-Rosen, D.G.1
Wu, H.2
Hahn, H.3
Osinska, T.4
Liron, J.N.5
Lorenz, A.6
Yatani, J.7
Robbins, G.W.8
Dorn II9
-
14
-
-
13044274187
-
Sustained in vivo cardiac protection by a rationally designed peptide that causes epsilon PKC translocation
-
Dorn II G.W., Souroujon M.C., Liron T., Chen C.H., Gray M.O., Zhou H.Z., Csukai M., Wu G., Lorenz J.N., Mochly-Rosen D. Sustained in vivo cardiac protection by a rationally designed peptide that causes epsilon PKC translocation. Proc. Natl. Acad. Sci. USA. 96:1999;12798-12803.
-
(1999)
Proc. Natl. Acad. Sci. USA
, vol.96
, pp. 12798-12803
-
-
Dorn II, G.W.1
Souroujon, M.C.2
Liron, T.3
Chen, C.H.4
Gray, M.O.5
Zhou, H.Z.6
Csukai, M.7
Wu, G.8
Lorenz, J.N.9
Mochly-Rosen, D.10
-
15
-
-
0034950242
-
Autoantibodies from mothers of children with congenital heart block directly crossreact and downregulate cardiac L-Type Ca channels
-
Qu Y.X., Chen L., Xiao G., Boutjdir M. Autoantibodies from mothers of children with congenital heart block directly crossreact and downregulate cardiac L-Type Ca channels. J. Mol. Cell. Cardiol. 33:2001;1153-1163.
-
(2001)
J. Mol. Cell. Cardiol.
, vol.33
, pp. 1153-1163
-
-
Qu, Y.X.1
Chen, L.2
Xiao, G.3
Boutjdir, M.4
-
16
-
-
0028030002
-
Increased protein kinase C and isozyme redistribution in pressure-overload cardiac hypertrophy in the rat
-
Gu X., Bishop S.P. Increased protein kinase C and isozyme redistribution in pressure-overload cardiac hypertrophy in the rat. Circ. Res. 75:1994;926-931.
-
(1994)
Circ. Res.
, vol.75
, pp. 926-931
-
-
Gu, X.1
Bishop, S.P.2
-
17
-
-
0038104862
-
Increased expression of protein kinase C isoforms in heart failure due to myocardial infarction
-
Wang J., Liu X., Sentex E., Takeda N., Dhalla N.S. Increased expression of protein kinase C isoforms in heart failure due to myocardial infarction. Am. J. Physiol. Heart Circ. Physiol. 284:2003;H2277-H2287.
-
(2003)
Am. J. Physiol. Heart Circ. Physiol.
, vol.284
-
-
Wang, J.1
Liu, X.2
Sentex, E.3
Takeda, N.4
Dhalla, N.S.5
-
18
-
-
0035893655
-
Role of tyrosine kinase activity in alpha-adrenergic inhibition of the beta-adrenergically regulated L-type Ca current in guinea-pig ventricular myocytes
-
Belevych A.E., Nulton-Persson A., Sims C., Harvey R.D. Role of tyrosine kinase activity in alpha-adrenergic inhibition of the beta-adrenergically regulated L-type Ca current in guinea-pig ventricular myocytes. J. Physiol. 537(Pt 3):2001;779-792.
-
(2001)
J. Physiol.
, vol.537
, Issue.PT 3
, pp. 779-792
-
-
Belevych, A.E.1
Nulton-Persson, A.2
Sims, C.3
Harvey, R.D.4
-
19
-
-
0035947740
-
Hypoxia alters the sensitivity of the L-type Ca channel to alpha-adrenergic receptor stimulation in the presence of beta-adrenergic receptor stimulation
-
Hool L.C. Hypoxia alters the sensitivity of the L-type Ca channel to alpha-adrenergic receptor stimulation in the presence of beta-adrenergic receptor stimulation. Circ. Res. 88:2001;1036-1043.
-
(2001)
Circ. Res.
, vol.88
, pp. 1036-1043
-
-
Hool, L.C.1
-
20
-
-
0029888788
-
Alpha 1-adrenergic inhibition of the beta-adrenergically activated Cl-current in guinea pig ventricular myocytes
-
Oleksa L.M., Hool L.C., Harvey R.D. Alpha 1-adrenergic inhibition of the beta-adrenergically activated Cl-current in guinea pig ventricular myocytes. Circ. Res. 78(6):1996;1090-1099.
-
(1996)
Circ. Res.
, vol.78
, Issue.6
, pp. 1090-1099
-
-
Oleksa, L.M.1
Hool, L.C.2
Harvey, R.D.3
-
21
-
-
0034710953
-
Inhibition of cardiac L-type Ca channels by PKC phosphorylation of two sites in the N-terminal domain
-
McHugh D., Sharp E.M., Scheuer T., Catterall W.A. Inhibition of cardiac L-type Ca channels by PKC phosphorylation of two sites in the N-terminal domain. Proc. Natl. Acad. Sci. USA. 97:2000;12334-12338.
-
(2000)
Proc. Natl. Acad. Sci. USA
, vol.97
, pp. 12334-12338
-
-
McHugh, D.1
Sharp, E.M.2
Scheuer, T.3
Catterall, W.A.4
-
22
-
-
0025255510
-
Phorbol ester and dioctanoylglycerol stimulate membrane association of PKC and have a negative inotropic effect mediated by changes in cytosolic Ca in adult rat cardiac myocytes
-
Capogrossi M.C., Kaku T., Filburn C.R., Pelto D.J., Hansford R.G., Spurgeon H.H., Lakatta E.G. Phorbol ester and dioctanoylglycerol stimulate membrane association of PKC and have a negative inotropic effect mediated by changes in cytosolic Ca in adult rat cardiac myocytes. Circ. Res. 66:1990;1143-1155.
-
(1990)
Circ. Res.
, vol.66
, pp. 1143-1155
-
-
Capogrossi, M.C.1
Kaku, T.2
Filburn, C.R.3
Pelto, D.J.4
Hansford, R.G.5
Spurgeon, H.H.6
Lakatta, E.G.7
-
23
-
-
0023902959
-
Phorbol ester increases Ca current and stimulates the effect of angiotensin II on cultured neonatal rat heart myocytes
-
Dosemesi A., Dhallan R.S., Cohen N.M., Lederer W.J., Rogers T.B. Phorbol ester increases Ca current and stimulates the effect of angiotensin II on cultured neonatal rat heart myocytes. Circ. Res. 62:1988;347-357.
-
(1988)
Circ. Res.
, vol.62
, pp. 347-357
-
-
Dosemesi, A.1
Dhallan, R.S.2
Cohen, N.M.3
Lederer, W.J.4
Rogers, T.B.5
-
24
-
-
0025874253
-
Expression of a constitutively activated mutant of the β-isozyme of PKC in cardiac myocytes stimulates the promotor of the β-myosin heavy chain isogene
-
Kariya K., Karns L.R., Simpson P.C. Expression of a constitutively activated mutant of the β-isozyme of PKC in cardiac myocytes stimulates the promotor of the β-myosin heavy chain isogene. J. Biol. Chem. 266:1990;10023-10026.
-
(1990)
J. Biol. Chem.
, vol.266
, pp. 10023-10026
-
-
Kariya, K.1
Karns, L.R.2
Simpson, P.C.3
-
25
-
-
0026349336
-
Regulation of cardiac gene expression during myocardial growth and hypertrophy: Molecular studies of an adaptive physiologic response
-
Chien K.R., Knowlton K.W., Chien S. Regulation of cardiac gene expression during myocardial growth and hypertrophy: molecular studies of an adaptive physiologic response. FASEB J. 5:1991;3037-3046.
-
(1991)
FASEB J.
, vol.5
, pp. 3037-3046
-
-
Chien, K.R.1
Knowlton, K.W.2
Chien, S.3
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