Morphological and Functional Alterations in Ventricular Myocytes from Male Transgenic Mice with Hypertrophic Cardiomyopathy

Circulation Research

Volumn 94, Issue 2, 2004, Pages 201-207

  Olsson, M Charlotte ^a,b   Palmer, Bradley M ^a,c   Stauffer, Brian L ^a   Leinwand, Leslie A ^a   Moore, Russell L ^a  

^a UNIVERSITY OF COLORADO   (United States)

^b UPPSALA UNIVERSITY   (Sweden)

^c UNIVERSITY OF VERMONT   (United States)

Author keywords

2,3 butanedione monoxime; Calcium; Heart failure; Relaxation

Indexed keywords

ACTIN BINDING PROTEIN; CELL PROTEIN; DIACETYL OXIME; MYOSIN HEAVY CHAIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CALCIUM CELL LEVEL; CELL FUNCTION; CELL STRUCTURE; CONTROLLED STUDY; DIASTOLE; FAMILIAL HYPERTROPHIC CARDIOMYOPATHY; GENE MUTATION; HEART LEFT VENTRICLE FAILURE; HEART MUSCLE CELL; HEART MUSCLE FIBROSIS; HEART VENTRICLE HYPERTROPHY; MALE; MOUSE; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; SARCOMERE LENGTH; SYSTOLE; TRANSGENIC MOUSE;

AGE FACTORS; AMINO ACID SUBSTITUTION; ANIMALS; CALCIUM; CARDIOMYOPATHY, HYPERTROPHIC, FAMILIAL; DIACETYL; DIASTOLE; GENE EXPRESSION; HEART VENTRICLES; MALE; MICE; MICE, TRANSGENIC; MICROSCOPY, ELECTRON; MODELS, ANIMAL; MUTATION, MISSENSE; MYOCARDIAL CONTRACTION; MYOCYTES, CARDIAC; MYOSIN HEAVY CHAINS; PHENOTYPE; PROTEIN ISOFORMS; SARCOMERES; SYSTOLE; VENTRICULAR DYSFUNCTION, LEFT; VENTRICULAR MYOSINS;

EID: 1042279539     PISSN: 00097330     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.RES.0000111521.40760.18     Document Type: Article

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