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Volumn 41, Issue 25, 2002, Pages 8093-8102
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Endogenous DNA lesions can inhibit the binding of the AP-1 (c-Jun) transcription factor
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Author keywords
[No Author keywords available]
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Indexed keywords
TRANSCRIPTION FACTOR;
DNA;
OXIDATION;
POLYMERS;
PROTEINS;
RNA;
BIOCHEMISTRY;
5 HYDROXYMETHYLURACIL;
CYTOSINE;
DNA GLYCOSYLTRANSFERASE;
GUANINE;
STRESS ACTIVATED PROTEIN KINASE;
TRANSCRIPTION FACTOR AP 1;
ARTICLE;
CONTROLLED STUDY;
DNA DAMAGE;
DNA REPAIR;
ENZYME ACTIVITY;
MISMATCH REPAIR;
NONHUMAN;
NUCLEIC ACID BASE SUBSTITUTION;
NUCLEOTIDE SEQUENCE;
OXIDATION;
PRIORITY JOURNAL;
PROTEIN BINDING;
PROTEIN DNA INTERACTION;
PROTEIN DOMAIN;
ADENINE;
BASE PAIR MISMATCH;
BINDING, COMPETITIVE;
CYTOSINE;
DNA DAMAGE;
DNA GLYCOSYLASES;
DNA-BINDING PROTEINS;
HUMANS;
N-GLYCOSYL HYDROLASES;
NUCLEIC ACID HETERODUPLEXES;
OLIGONUCLEOTIDES;
PENTOXYL;
PROTEIN BINDING;
PROTO-ONCOGENE PROTEINS C-JUN;
THYMINE;
TRANSCRIPTION FACTOR AP-1;
URACIL;
URACIL-DNA GLYCOSIDASE;
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EID: 0037172798
PISSN: 00062960
EISSN: None
Source Type: Journal
DOI: 10.1021/bi012180a Document Type: Article |
Times cited : (35)
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References (83)
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