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Volumn 22, Issue 1, 2001, Pages 179-186
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Excessive base excision repair of 5-hydroxymethyluracil from DNA induces apoptosis in Chinese hamster V79 cells containing mutant p53
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Author keywords
[No Author keywords available]
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Indexed keywords
3 AMINOBENZAMIDE;
5 HYDROXYMETHYLDEOXYURIDINE;
5 HYDROXYMETHYLURACIL;
BROXURIDINE;
DNA;
DNA NUCLEOTIDYLEXOTRANSFERASE;
DOUBLE STRANDED DNA;
FLUORESCEIN ISOTHIOCYANATE;
MUTANT PROTEIN;
NICOTINAMIDE ADENINE DINUCLEOTIDE;
POLY(ADENOSINE DIPHOSPHATE RIBOSE);
PROTEIN P53;
ANIMAL CELL;
APOPTOSIS;
ARTICLE;
CELL CYCLE G2 PHASE;
CELL CYCLE S PHASE;
CELL MUTANT;
CELL STRAIN V 79;
CHINESE HAMSTER;
CONTROLLED STUDY;
DNA DAMAGE;
DNA REPAIR;
DNA STRAND BREAKAGE;
EXCISION REPAIR;
FLOW CYTOMETRY;
IMMUNOCHEMISTRY;
MOLECULAR WEIGHT;
NONHUMAN;
NUCLEOSOME;
PHASE CONTRAST MICROSCOPY;
PRIORITY JOURNAL;
PULSED FIELD GEL ELECTROPHORESIS;
ANIMALS;
APOPTOSIS;
BROMODEOXYURIDINE;
CELL CYCLE;
CELL MEMBRANE;
CRICETINAE;
CRICETULUS;
DNA;
DNA DAMAGE;
DNA NUCLEOTIDYLEXOTRANSFERASE;
DNA REPAIR;
FIBROBLASTS;
FLUORESCEIN-5-ISOTHIOCYANATE;
G2 PHASE;
GENES, P53;
HL-60 CELLS;
HUMANS;
LUNG;
MICE;
MUTATION;
PENTOXYL;
PHOSPHATIDYLSERINES;
THYMIDINE;
TUMOR SUPPRESSOR PROTEIN P53;
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EID: 0035136043
PISSN: 01433334
EISSN: None
Source Type: Journal
DOI: 10.1093/carcin/22.1.179 Document Type: Article |
Times cited : (8)
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References (47)
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