Reduction of Ito causes hypertrophy in neonatal rat ventricular myocytes

Circulation Research

Volumn 90, Issue 5, 2002, Pages 578-585

  Kassiri, Zamaneh ^a   Zobel, Carsten ^a   Nguyen, The Tin T ^a   Molkentin, Jeffery D ^b   Backx, Peter H ^a  

^a UNIVERSITY OF TORONTO   (Canada)

^b CINCINNATI CHILDREN'S HOSPITAL MEDICAL CENTER   (United States)

Author keywords

Ca2+ i; Cardiac myocyte; Hypertrophy; Transient outward K+ current

Indexed keywords

CALCINEURIN; CALCINEURIN INHIBITOR; CALCIUM ION; POTASSIUM CHANNEL; POTASSIUM ION; TOXIN;

ANIMAL TISSUE; ARTICLE; CELL MEMBRANE; CONTROLLED STUDY; ENZYME ACTIVITY; GENE EXPRESSION; GENE OVEREXPRESSION; HEART ELECTROPHYSIOLOGY; HEART MUSCLE CELL; HEART MUSCLE POTENTIAL; HEART VENTRICLE HYPERTROPHY; HYPERTROPHY; NONHUMAN; POTASSIUM CURRENT; PRIORITY JOURNAL; RAT; SIGNAL TRANSDUCTION; TRANSGENE;

ACTION POTENTIALS; AMINO ACID SUBSTITUTION; ANIMALS; ANIMALS, NEWBORN; CALCINEURIN; CALCIUM; CALCIUM CHANNEL BLOCKERS; CARDIOMEGALY; CARRIER PROTEINS; CELLS, CULTURED; GENE EXPRESSION; GENES, DOMINANT; HEART VENTRICLES; KV1.4 POTASSIUM CHANNEL; MUTAGENESIS, SITE-DIRECTED; MYOCARDIUM; PATCH-CLAMP TECHNIQUES; POTASSIUM; POTASSIUM CHANNEL BLOCKERS; POTASSIUM CHANNELS; POTASSIUM CHANNELS, VOLTAGE-GATED; RATS; RATS, SPRAGUE-DAWLEY; SHAL POTASSIUM CHANNELS; SPIDER VENOMS; TRANSGENES;

EID: 0037155771     PISSN: 00097330     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.RES.0000012223.86441.A1     Document Type: Article

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