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Volumn 277, Issue 38, 2002, Pages 35305-35313
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Protein kinase C-ε promotes survival of lung cancer cells by suppressing apoptosis through dysregulation of the mitochondrial caspase pathway
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Author keywords
[No Author keywords available]
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Indexed keywords
ACTIVATION ANALYSIS;
CELLS;
CHEMOTHERAPY;
DNA;
DRUG PRODUCTS;
GENES;
PULMONARY DISEASES;
TUMORS;
APOPTOSIS;
ENZYMES;
CASPASE;
CYTOCHROME C;
DOXORUBICIN;
ETOPOSIDE;
PROTEIN KINASE C EPSILON;
APOPTOSIS;
ARTICLE;
CANCER CELL;
CELL CYCLE G1 PHASE;
CELL DEATH;
CELL SURVIVAL;
ENZYME ACTIVATION;
GENE INACTIVATION;
HUMAN;
HUMAN CELL;
LUNG CANCER;
LUNG NON SMALL CELL CANCER;
MITOCHONDRIAL MEMBRANE;
NORTHERN BLOTTING;
PRIORITY JOURNAL;
TRANSCRIPTION REGULATION;
ANTINEOPLASTIC AGENTS;
APOPTOSIS;
CARCINOMA, SMALL CELL;
CASPASES;
CELL SURVIVAL;
DNA, ANTISENSE;
DNA, COMPLEMENTARY;
DRUG RESISTANCE, NEOPLASM;
ENZYME ACTIVATION;
ETOPOSIDE;
GENE EXPRESSION REGULATION, ENZYMOLOGIC;
HUMANS;
ISOENZYMES;
LUNG NEOPLASMS;
MITOCHONDRIA;
PHENOTYPE;
PROTEIN KINASE C;
PROTEIN KINASE C-EPSILON;
TRANSCRIPTION, GENETIC;
TUMOR CELLS, CULTURED;
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EID: 0037144492
PISSN: 00219258
EISSN: None
Source Type: Journal
DOI: 10.1074/jbc.M201460200 Document Type: Article |
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Times cited : (96)
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References (44)
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