SCOPUS 정보 검색 플랫폼

Volumn 277, Issue 16, 2002, Pages 13371-13374

Loss of p53 compensates for αv-integrin function in retinal neovascularization

(7) Strömblad, Staffan a Fotedar, Arun a Brickner, Howard b Theesfeld, Chandra c De Diaz, Edith Aguilar d Friedlander, Martin e Cheresh, David A f

a KAROLINSKA INSTITUTE (Sweden)

b SÖDERTÖRN UNIVERSITY (Sweden)

c SIDNEY KIMMEL CANCER CENTER (United States)

d Department of Cell Biology ^* (United States)

e SCRIPPS RESEARCH INSTITUTE (United States)

f KAROLINSKA UNIVERSITY HOSPITAL (Sweden)

Author keywords

[No Author keywords available]

Indexed keywords

BIODIVERSITY; BLOOD VESSELS; CARDIOVASCULAR SYSTEM;

NEOVASCULARIZATION;

BIOCHEMISTRY;

ALPHA5 INTEGRIN; PROTEIN P21; PROTEIN P53;

ANGIOGENESIS; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; GENE CONTROL; GENE LOSS; GENE REPRESSION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEPLETION; PROTEIN FUNCTION; PROTEIN INDUCTION; RETINA NEOVASCULARIZATION; TUMOR SUPPRESSOR GENE;

ANIMALS; ANTIGENS, CD; BLOTTING, WESTERN; CYCLIN-DEPENDENT KINASE INHIBITOR P21; CYCLINS; DOSE-RESPONSE RELATIONSHIP, DRUG; GENOTYPE; HETEROZYGOTE; INTEGRIN ALPHAV; MICE; MICE, KNOCKOUT; MICROSCOPY, FLUORESCENCE; MODELS, BIOLOGICAL; NEOVASCULARIZATION, PHYSIOLOGIC; PROTEIN BINDING; RETINA; SIGNAL TRANSDUCTION; TUMOR SUPPRESSOR PROTEIN P53;

ANIMALIA; MURINAE;

EID: 0037134486 PISSN: 00219258 EISSN: None Source Type: Journal
DOI: 10.1074/jbc.C200044200 Document Type: Article

Times cited : (27)

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