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Oncogene

Volumn 21, Issue 22, 2002, Pages 3525-3531

Loss of p19ARF enhances the defects of Mdm2 overexpression in the mammary gland

(2) Foster, Carolyn J a Lozano, Guillermina a

a UNIVERSITY OF TEXAS MD ANDERSON CANCER CENTER (United States)

Author keywords

Cell cycle; p53; Proliferation

Indexed keywords

PROTEIN MDM2; PROTEIN P19; PROTEIN P53;

ANIMAL CELL; APOPTOSIS; ARTICLE; BREAST CANCER; CELL DIVISION; CELL PROLIFERATION; CYTOKINESIS; GENE INACTIVATION; GENE OVEREXPRESSION; GENE TARGETING; HETEROZYGOSITY LOSS; MOUSE; NONHUMAN; PRIORITY JOURNAL; TRANSCRIPTION REGULATION; TRANSGENE; TUMOR GROWTH; TUMOR SUPPRESSOR GENE;

ANIMALS; CELL CYCLE; CYCLIN-DEPENDENT KINASE INHIBITOR P16; DNA REPLICATION; EPITHELIAL CELLS; FEMALE; GIANT CELLS; LACTATION; MAMMARY GLANDS, ANIMAL; MICE; MICE, KNOCKOUT; MICE, TRANSGENIC; NUCLEAR PROTEINS; PHENOTYPE; PREGNANCY; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-MDM2; TUMOR SUPPRESSOR PROTEIN P14ARF;

ANIMALIA; MURINAE; MUS MUSCULUS;

EID: 0037118583 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1205441 Document Type: Article

Times cited : (10)

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