Inhibition of mitochondrial protein synthesis results in increased endothelial cell susceptibility to nitric oxide-induced apoptosis

Proceedings of the National Academy of Sciences of the United States of America

Volumn 99, Issue 10, 2002, Pages 6643-6648

  Ramachandran, Anup ^a   Moellering, Douglas R ^a   Ceaser, Erin ^a   Shiva, Sruti ^a   Xu, Jun ^a   Darley Usmar, Victor ^a,b  

^a University of Alabama at Birmingham   (United States)

^b UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CHLORAMPHENICOL; CYTOCHROME C; MITOCHONDRIAL DNA; NITRIC OXIDE; PROTEIN;

ANIMAL CELL; APOPTOSIS; ARTICLE; CYTOTOXICITY; ENDOTHELIUM CELL; GENE MUTATION; MITOCHONDRION; NONHUMAN; PRIORITY JOURNAL; PROTEIN SYNTHESIS INHIBITION; SIGNAL TRANSDUCTION;

ADENOSINE TRIPHOSPHATE; ALKENES; ANIMALS; AORTA; APOPTOSIS; CASPASE 3; CASPASES; CATTLE; CELLS, CULTURED; CHLORAMPHENICOL; CYTOCHROME C GROUP; ELECTRON TRANSPORT; ELECTRON TRANSPORT COMPLEX II; ENDOTHELIUM, VASCULAR; ENERGY METABOLISM; MITOCHONDRIA; MULTIENZYME COMPLEXES; NITRIC OXIDE; NITRIC OXIDE DONORS; OXIDOREDUCTASES; PROTEIN BIOSYNTHESIS; PROTEIN SYNTHESIS INHIBITORS; REACTIVE OXYGEN SPECIES; SUCCINATE DEHYDROGENASE;

ANIMALIA; BOVINAE;

EID: 0037076412     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.102019899     Document Type: Article

References (41)