Increased Ras expression and caspase-independent neuroblastoma cell death: Possible mechanism of spontaneous neuroblastoma regression

Journal of the National Cancer Institute

Volumn 94, Issue 5, 2002, Pages 358-368

  Kitanaka, Chifumi ^a   Kato, Keisuke ^b   Ijiri, Rieko ^b   Sakurada, Kaori ^a   Tomiyama, Arata ^a   Noguchi, Kohji ^a   Nagashima, Yohji ^c   Nakagawara, Akira ^d   Momoi, Takashi ^e   Toyoda, Yasunori ^b   Kigasawa, Hisato ^b   Nishi, Toshiji ^b   Shirouzu, Mikako ^f   Yokoyama, Shigeyuki ^f,g   Tanaka, Yukichi ^b   Kuchino, Yoshiyuki ^a,h  

^a NATIONAL CANCER CENTER RESEARCH INSTITUTE   (Japan)

^b KANAGAWA CHILDREN'S MEDICAL CENTER   (Japan)

^c YOKOHAMA CITY UNIVERSITY   (Japan)

^d CHIBA CANCER CENTER   (Japan)

^e NATIONAL INSTITUTE OF NEUROSCIENCE   (Japan)

^f RIKEN YOKOHAMA INSTITUTE   (Japan)

^g UNIVERSITY OF TOKYO   (Japan)

^h JAPAN SCIENCE AND TECHNOLOGY AGENCY   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE 3; DNA FRAGMENT; NERVE GROWTH FACTOR RECEPTOR; RAS PROTEIN;

ARTICLE; AUTOPHAGY; CANCER SCREENING; CANCER STAGING; CELL DEATH; CELL DEGENERATION; CELL STRUCTURE; CLINICAL ARTICLE; CONTROLLED STUDY; ELECTRON MICROSCOPY; ENZYME ACTIVATION; GENETIC TRANSFECTION; HUMAN; HUMAN TISSUE; IMMUNOHISTOCHEMISTRY; MICROSCOPY; NEUROBLASTOMA; NEUROBLASTOMA CELL; ONCOGENE RAS; PRIORITY JOURNAL; PROGNOSIS; PROTEIN EXPRESSION; TUMOR BIOPSY; TUMOR REGRESSION;

EID: 0037029077     PISSN: 00278874     EISSN: None     Source Type: Journal    
DOI: 10.1093/jnci/94.5.358     Document Type: Article

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