Impaired hypoxic tolerance and altered protein binding of NADH in presymptomatic APP23 transgenic mice

Neuroscience

Volumn 114, Issue 2, 2002, Pages 285-289

  Buchner M ^a   Huber, R ^a   Sturchler Pierrat, C ^b   Staufenbiel, M ^b   Riepe, Matthias W ^a  

^a UNIVERSITY OF ULM   (Germany)

^b NOVARTIS PHARMA AG   (Switzerland)

Author keywords

Alzheimer; Hypoxia; NADH; Preconditioning; Spectrum

Indexed keywords

AMYLOID; PROTEIN; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE;

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN REGION; BRAIN SLICE; CONTROLLED STUDY; ENERGY METABOLISM; FLUORESCENCE SPECTROSCOPY; HIPPOCAMPUS; HYPOXIA; MICROENVIRONMENT; MOUSE; MOUSE MUTANT; NONHUMAN; OXIDATION; PRIORITY JOURNAL; PROTEIN BINDING; PYRAMIDAL NERVE CELL; TRANSGENIC MOUSE;

ACTION POTENTIALS; ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN PRECURSOR; ANIMALS; DISEASE MODELS, ANIMAL; ENERGY METABOLISM; FEMALE; HIPPOCAMPUS; HYPOXIA, BRAIN; ISCHEMIC PRECONDITIONING; MALE; MICE; MICE, TRANSGENIC; NAD; NEURONS; NITRO COMPOUNDS; PROPIONIC ACIDS; PROTEIN BINDING; RECOVERY OF FUNCTION; SPECTRUM ANALYSIS;

EID: 0036774859     PISSN: 03064522     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0306-4522(02)00280-4     Document Type: Article

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