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Volumn 35, Issue 1, 2002, Pages 1-5

Follistatin overexpression in rodent liver tumors: A possible mechanism to overcome activin growth control

Author keywords

Apoptosis; Gene expression; Hepatocarcinogenesis; Liver cancer

Indexed keywords

ACTIVIN; CARCINOGEN; DNA; FOLLISTATIN; MESSENGER RNA;

EID: 0036735319     PISSN: 08991987     EISSN: None     Source Type: Journal    
DOI: 10.1002/mc.10068     Document Type: Article
Times cited : (32)

References (41)
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    • Phillips, D.J.1
  • 25
    • 0031817014 scopus 로고    scopus 로고
    • Simultaneous quantitation of substance P-encoding preprotachykinin alternatively spliced mRNAs and substance P receptor NK-1 mRNA by an RNase protection assay
    • (1998) Brain Res Protoc , vol.2 , pp. 133-140
    • Gautreau, A.1    Kerdelhue, B.2
  • 34
    • 0033279446 scopus 로고    scopus 로고
    • Differential response to exogenous and endogenous activin in a human ovarian teratocarcinoma-derived cell line (PA-1): Regulation by cell surface follistatin
    • (1999) Endocrinology , vol.140 , pp. 2463-2470
    • Delbaere, A.1    Sidis, Y.2    Schneyer, A.L.3
  • 41
    • 0033030308 scopus 로고    scopus 로고
    • Transforming growth factor-β signaling and cirrhosis
    • (1999) Hepatology , vol.29 , pp. 1909-1910
    • Wrana, J.L.1


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.