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Volumn 35, Issue 1, 2002, Pages 1-5
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Follistatin overexpression in rodent liver tumors: A possible mechanism to overcome activin growth control
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Author keywords
Apoptosis; Gene expression; Hepatocarcinogenesis; Liver cancer
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Indexed keywords
ACTIVIN;
CARCINOGEN;
DNA;
FOLLISTATIN;
MESSENGER RNA;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
CANCER GRADING;
CANCER STAGING;
CONTROLLED STUDY;
DNA SYNTHESIS;
GROWTH REGULATION;
HISTOPATHOLOGY;
HOMEOSTASIS;
HUMAN;
HUMAN CELL;
LIVER ADENOMA;
LIVER CELL CARCINOMA;
LIVER TUMOR;
MALE;
MOUSE;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN BINDING;
PROTEIN EXPRESSION;
RAT;
RIBONUCLEASE PROTECTION ASSAY;
RNA ANALYSIS;
RODENT;
SIGNAL TRANSDUCTION;
ACTIVINS;
ADENOMA;
ALTERNATIVE SPLICING;
ANIMALS;
BLOTTING, WESTERN;
CARCINOMA, HEPATOCELLULAR;
CELL DIVISION;
DIETHYLNITROSAMINE;
FOLLISTATIN;
GENE EXPRESSION REGULATION, NEOPLASTIC;
HUMANS;
LIVER NEOPLASMS;
MALE;
MICE;
MICE, INBRED STRAINS;
NAFENOPIN;
PHENOBARBITAL;
RATS;
REFERENCE VALUES;
RNA, MESSENGER;
TRANSFORMING GROWTH FACTOR ALPHA;
TUMOR CELLS, CULTURED;
ANIMALIA;
RODENTIA;
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EID: 0036735319
PISSN: 08991987
EISSN: None
Source Type: Journal
DOI: 10.1002/mc.10068 Document Type: Article |
Times cited : (32)
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References (41)
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