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Volumn 67, Issue 3, 2002, Pages 789-794
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Src tyrosine kinase-induced loss of luteinizing hormone responsiveness is via a Ras-dependent, phosphatidylinositol-3-kinase independent pathway
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Author keywords
Growth factors; Kinases; Leydig cells; Signal transduction
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Indexed keywords
8 BROMO CYCLIC AMP;
CHOLERA TOXIN;
CYCLIC AMP;
CYCLIC AMP DEPENDENT PROTEIN KINASE;
GROWTH FACTOR;
LUTEINIZING HORMONE;
PHOSPHATIDYLINOSITOL 3 KINASE;
PROTEIN TYROSINE KINASE;
RAS PROTEIN;
WORTMANNIN;
ANIMAL CELL;
ARTICLE;
CONTROLLED STUDY;
ENZYME ACTIVATION;
ENZYME INDUCTION;
ENZYME INHIBITION;
ENZYME MECHANISM;
HORMONE ACTION;
LEYDIG CELL;
NONHUMAN;
PRIORITY JOURNAL;
PROGESTERONE RELEASE;
PROTEIN EXPRESSION;
SIGNAL TRANSDUCTION;
STEROID RELEASE;
STEROIDOGENESIS;
TEMPERATURE SENSITIVITY;
1-PHOSPHATIDYLINOSITOL 3-KINASE;
ANDROSTADIENES;
ENZYME ACTIVATION;
ENZYME INHIBITORS;
GENE EXPRESSION;
GUANOSINE DIPHOSPHATE;
GUANOSINE TRIPHOSPHATE;
LEYDIG CELL TUMOR;
LUTEINIZING HORMONE;
MUTAGENESIS;
PROGESTERONE;
RAS PROTEINS;
SIGNAL TRANSDUCTION;
SRC-FAMILY KINASES;
TRANSFECTION;
TUMOR CELLS, CULTURED;
ANIMALIA;
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EID: 0036720834
PISSN: 00063363
EISSN: None
Source Type: Journal
DOI: 10.1095/biolreprod.101.000976 Document Type: Article |
Times cited : (9)
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References (31)
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