Interstitial inflammation, sodium retention, and the pathogenesis of nephrotic edema: A unifying hypothesis

Kidney International

Volumn 62, Issue 4, 2002, Pages 1379-1384

  Rodríguez Iturbe, Bernardo ^a   Herrera Acosta, Jaime ^b   Johnson, Richard J ^c  

^a HOSPITAL UNIVERSITARIO DE MARACAIBO   (Venezuela)

^b INSTITUTO NACIONAL DE CARDIOLOGÍA IGNACIO CHÁVEZ   (Mexico)

^c BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

Glomerulonephritis; Hypoalbuminemia; Interstitial cells; Nephrotic syndrome; Renin angiotensin

Indexed keywords

MONOCYTE CHEMOTACTIC PROTEIN 1; MYCOPHENOLIC ACID 2 MORPHOLINOETHYL ESTER; SARALASIN;

ARTICLE; BLOOD VOLUME; DRUG EFFECT; EDEMA; GLOMERULUS FILTRATION RATE; HEMODYNAMICS; INTERSTITIUM; NEPHROSIS; NONHUMAN; ONCOTIC PRESSURE; PATHOPHYSIOLOGY; PHENOTYPE; PRIORITY JOURNAL; PROTEINURIA; SODIUM LOAD; SODIUM RETENTION;

EID: 0036378195     PISSN: 00852538     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1523-1755.2002.00561.x     Document Type: Article

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