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Volumn 283, Issue 3 52-3, 2002, Pages

Enhancement of closed-state inactivation in long QT syndrome sodium channel mutation ΔKPQ

Author keywords

Anthopleurin; Gating current; Heart; Nav1.5 channel; Site 3

Indexed keywords

AMINO ACID; ANTHOPLEURIN A; GLUTAMINE; LYSINE; PROLINE; SODIUM CHANNEL;

EID: 0036351760     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.00097.2002     Document Type: Article
Times cited : (7)

References (41)
  • 7
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  • 8
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    • Structure and function of voltage-sensitive ion channels
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    • Catterall, W.A.1
  • 13
    • 25044474835 scopus 로고    scopus 로고
    • Augmentation in closed state to inactivated state kinetics in the LQT sodium channel mutation, DeltaKPQ, by site-3 toxins
    • (2002) Biophys J , vol.82
    • Chen, T.1    Sheets, M.F.2
  • 17
  • 21
    • 0030885219 scopus 로고    scopus 로고
    • Sodium channel inactivation is altered by substitution of voltage sensor positive charges
    • published erratum appears in J Gen Physiol 110: 765, 1997
    • (1997) J Gen Physiol , vol.110 , pp. 403-413
    • Kontis, K.J.1    Goldin, A.L.2
  • 31
    • 0028811042 scopus 로고
    • Voltage-dependent open-state inactivation of cardiac sodium channels: Gating currents studies with Anthopleurin-A toxin
    • (1995) J Gen Physiol , vol.106 , pp. 617-640
    • Sheets, M.F.1    Hanck, D.A.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.