Brugada syndrome: Why are there multiple answers to a simple question?

Circulation

Volumn 104, Issue 25, 2001, Pages 3017-3019

  Brugada, Ramon ^a   Roberts, Robert ^a  

^a BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

Arrhythmia; Bundle branch block; Editorials; Fibrillation; Genetics

Indexed keywords

BRUGADA SYNDROME; EDITORIAL; GENE MUTATION; GENETIC DISORDER; HUMAN; MEDICAL RESEARCH; PRIORITY JOURNAL; ST SEGMENT ELEVATION;

EID: 0035909918     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/circ.104.25.3017     Document Type: Editorial

References (19)

1. Right bundle branch block, persistent ST segment elevation and sudden cardiac death: A distinct clinical and electrocardiographic syndrome
2. Right bundle branch block and ST segment elevation in leads V1-V3: A marker for sudden death in patients with no demonstrable structural heart disease
3. Arrhythmogenic marker for the sudden unexplained death syndrome in Thai men
4. Sudden death among Southeast Asian refugees: An unexplained nocturnal phenomenon
5. Brugada syndrome and sudden cardiac death in children
6. The Brugada syndrome: Ionic basis and arrhythmia mechanisms
7. Genetic basis and molecular mechanisms for idiopathic ventricular fibrillation
8. Ionic mechanisms responsible for the electrocardiographic phenotype of the Brugada syndrome are temperature dependent
9. Sudden death in high-risk family members: Brugada syndrome
10. The prognostic value of electrophysiologic investigation in Brugada syndrome
11. Autonomic and antiarrhythmic modulation of ST segment elevation in patients with Brugada syndrome
12. Sodium channel blockers identify risk for sudden death in patients with ST segment elevation and right bundle branch block but structurally normal hearts
13. Clinical and genetic heterogeneity of right bundle branch block and ST-segment elevation syndrome: A prospective evaluation of 52 families
14. Cardiac conduction defects associate with mutations in SCN5A
15. Multiple mechanisms in the long-QT syndrome: Current knowledge, gaps and future directions
16. + channel mutation causing both long-QT and Brugada syndrome
17. Novel SCN5A mutation leading either to isolated cardiac conduction defect or Brugada syndrome in a large French family
18. The power of genome-wide association studies of complex disease genes: Statistical limitations of indirect approaches using SNP markers
19. 3