Reduced soluble CD14 levels in amniotic fluid and breast milk are associated with the subsequent development of atopy, eczema, or both

Journal of Allergy and Clinical Immunology

Volumn 109, Issue 5, 2002, Pages 858-866

  Jones, Catherine A ^a   Holloway, Judith A ^a   Popplewell, Eleanor J ^a   Diaper, Norma D ^a   Holloway, John W ^a   Vance, Gillian H S ^a   Warner, Jill A ^a   Warner, John O ^a  

^a UNIVERSITY OF SOUTHAMPTON   (United Kingdom)

Author keywords

Amniotic fluid; Breast milk; Eczema; Soluble CD14

Indexed keywords

CD14 ANTIGEN; LIPOPOLYSACCHARIDE; TOLL LIKE RECEPTOR;

AMNION FLUID; ARTICLE; ATOPY; BREAST MILK; CHILD; DISEASE ASSOCIATION; ECZEMA; ENZYME LINKED IMMUNOSORBENT ASSAY; FETUS DEVELOPMENT; GENE EXPRESSION; HUMAN; IMMUNOREACTIVITY; INFANT; INTESTINE; NEWBORN; PERINATAL PERIOD; PRIORITY JOURNAL; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SKIN;

EID: 0036251123     PISSN: 00916749     EISSN: None     Source Type: Journal    
DOI: 10.1067/mai.2002.123535     Document Type: Article

References (32)

1. Family size, infection and atopy: The first decade of the hygiene hypothesis
2. Atopy versus infectious diseases in childhood: A question of balance
3. Microflora-associated characteristics in faeces from allergic and non-allergic infants
4. The intestinal microflora in allergic Estonian and Swedish 2-year-old children
5. Reduced risk of hayfever and asthma among children of farmers
6. Austrian children living on a farm have less hayfever, asthma and allergic sensitization
7. Farm environment in childhood prevents the development of allergies
8. Early childhood infection and atopic disorder
9. Antibiotic use in early childhood and the development of asthma
10. Probiotics in primary prevention of atopic disease: A randomised placebo-controlled trial
11. The requirement of intestinal bacterial flora for the development of an IgE production system susceptible to oral tolerance induction
12. Transplacental priming of the human immune system to environmental allergens: Universal skewing of initial T cell responses toward the Th2 cytokine profile
13. Development of allergen-specific T cell memory in atopic and normal children
14. The immune system evolved to discriminate infectious non-self from non-infectious self
15. The monocytic differentiation antigen, CD14, is anchored to the cell by phosphatidyl inositol linkage
16. Toll-like receptor 4 imparts ligand-specific recognition of bacterial lipopolysaccharide
17. TLR4: Central component of the sole mammalian LPS sensor
18. Soluble CD14 participates in the response of cells to lipopolysaccharide
19. A polymorphism in the 5′ flanking region of the CD14 gene is associated with circulating soluble CD14 levels and with total serum immunoglobulin E
20. Association of a promoter polymorphism of the CD14 gene and atopy
21. Primary prevention of asthma with Cetirizine in infants with atopic dermatitis. First results of ETAC: A double blind randomised placebo-controlled trial
22. Variations in serum soluble IL-2 receptor concentration
23. Early exposure to house dust mite and cat allergens and development of childhood asthma: A cohort study
24. Lipopolysaccharide binding protein and soluble CD14 receptor protein in amniotic fluid and cord blood in patients at term
25. Human B cell function is regulated by interaction with soluble CD14: Opposite effects on IgG1 and IgE production
26. The development of gut associated lymphoid tissue in the terminal ileum of fetal human intestine
27. Lipopolysaccharide induces physical proximity between CD14 and toll-like receptor 4 (TLR 4) prior to nuclear translocation of NF-κB
28. Differential roles of TLR2 and TLR4 in recognition of gram-negative and gram-positive bacterial cell wall components
29. Costimulatory molecules in the developing human gastrointestinal tract: A pathway for fetal allergen priming
30. Innate recognition of bacteria in human milk is mediated by a milk-derived highly expressed pattern recognition receptor, soluble CD14
31. Soluble CD14 mediates efflux of phospholipids from cells
32. Essential fatty acid metabolism and its modification in atopic eczema