Immune reconstitution after successful treatment with protease inhibitor-based and protease inhibitor-sparing antiretroviral regimens [1]

Journal of Acquired Immune Deficiency Syndromes

Volumn 29, Issue 5, 2002, Pages 544-545

  Smith, Kimberly Y ^a   Steffens, Carolyn M ^a   Truckenbrod, Allison ^a   Landay, Alan ^a   Al Harthi, Lena ^a  

^a RUSH UNIVERSITY MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIRETROVIRUS AGENT; CD38 ANTIGEN; HLA DR ANTIGEN; INDINAVIR; NELFINAVIR; PROTEINASE INHIBITOR; RITONAVIR; RNA DIRECTED DNA POLYMERASE INHIBITOR;

ACQUIRED IMMUNE DEFICIENCY SYNDROME; ADULT; ANTIGEN EXPRESSION; CLINICAL ARTICLE; DISEASE COURSE; HUMAN; HUMAN IMMUNODEFICIENCY VIRUS 1; HUMAN IMMUNODEFICIENCY VIRUS INFECTION; IMMUNE RESPONSE; IMMUNOSTIMULATION; LETTER; LYMPHOCYTE COUNT; PRIORITY JOURNAL; T LYMPHOCYTE SUBPOPULATION; VIRUS LOAD;

EID: 0036236334     PISSN: 15254135     EISSN: None     Source Type: Journal    
DOI: 10.1097/00126334-200204150-00017     Document Type: Letter

References (10)

1. Efavirenz plus indinavir, and indinavir plus zidovudine and lamivudine in the treatment of HIV-1 infection in adults
2. Immunologic responses associated with 12 weeks of combination antiretroviral therapy consisting of zidovudine, lamivudine, and ritonavir: Results of AIDS Clinical Trials Groups Protocol 315
3. Immune reconstitution in the first year of potent antiretroviral therapy and its relationship to virologic response
4. Detection of T cell receptor circles (TRECs) as biomarkers for de novo T cell synthesis using a quantitative polymerase chain reaction enzyme-linked immunosorbent assay (PCR-ELISA)
5. Changes in thymic function with age and during the treatment of HIV infection
6. CD8+, CD38+ lymphocyte percent: A useful immunological marker for monitoring HIV-1-infected patients
7. Elevated CD38 antigen expression on CD8+ T cells is a stronger marker for the risk of chronic HIV disease progression to AIDS and death in the Multicenter AIDS Cohort Study than CD4+ count, soluble immune activation markers, or combinations of HLA-DR and CD38+ expression
8. CD8+ T-lymphocyte Activation in HIV-1 disease reflects an aspect of pathogenesis distinct from viral burden and immunodeficiency
9. Cellular restoration in HIV-infected persons treated with abacavir and a protease inhibitor: Age inversely predicts naive CD4 cell count increase
10. Increased cell division but not thymic dysfunction rapidly affects the T-cell receptor excision circle content of the naive T-cell population in HIV-1 infection