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Volumn 88, Issue 3, 2002, Pages 341-350
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Electrophysiological, anatomical and histological remodeling of the heart to AV block enhances susceptibility to arrhythmogenic effects of QT-prolonging drugs
a a a a a a a a |
Author keywords
AV block; Holter ECG; Long QT syndrome; Monophasic action potential; MR image
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Indexed keywords
ANTIARRHYTHMIC AND ARRHYTHMIA-INDUCING AGENTS;
CISAPRIDE;
COLLAGEN FIBER;
HEPARIN CALCIUM;
ACTION POTENTIAL;
ACUTE DISEASE;
ADAPTATION;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ARTICLE;
ATRIOVENTRICULAR BLOCK;
BLOOD CLOTTING DISORDER;
CARDIOVASCULAR RISK;
CHRONIC DISEASE;
COMPARATIVE STUDY;
CONTROLLED STUDY;
DISEASE COURSE;
DISEASE PREDISPOSITION;
DOG;
DOSE RESPONSE;
DRUG EFFECT;
DRUG INDUCED DISEASE;
ELECTRON MICROSCOPY;
EXTRACELLULAR SPACE;
FEMALE;
HEART DEPOLARIZATION;
HEART ELECTROPHYSIOLOGY;
HEART HYPERTROPHY;
HEART MUSCLE REFRACTORY PERIOD;
HEART RIGHT VENTRICLE HYPERTROPHY;
HEART VENTRICLE REMODELING;
HEART VENTRICLE SEPTUM;
HEART VENTRICLE WALL;
HEART WEIGHT;
HISTOPATHOLOGY;
HOLTER MONITORING;
LONG QT SYNDROME;
MALE;
MICROPHOTOGRAPHY;
NONHUMAN;
NUCLEAR MAGNETIC RESONANCE IMAGING;
PATHOLOGICAL ANATOMY;
REENTRY ARRHYTHMIA;
TORSADE DES POINTES;
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EID: 0036213772
PISSN: 00215198
EISSN: None
Source Type: Journal
DOI: 10.1254/jjp.88.341 Document Type: Article |
Times cited : (56)
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References (29)
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