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Volumn 4, Issue 1, 2002, Pages 15-23
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Antihistamine terfenadine inhibits calcium influx, cGMP formation, and NMDA receptor-dependent neurotoxicity following activation of L-type voltage sensitive calcium channels
a a b c
b
Uniformed Services University of the Health Sciences and the Walter Reed National Military Medical Center
(United States)
c
NONE
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Author keywords
cGMP; Cultured cerebellar neurons; Excitotoxicity; Terfenadine; Voltage sensitive calcium channels
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Indexed keywords
1,4 DIHYDRO 2,6 DIMETHYL 5 NITRO 4 [2 (TRIFLUOROMETHYL)PHENYL] 3 PYRIDINECARBOXYLIC ACID METHYL ESTER;
CALCIUM CHANNEL L TYPE;
CALCIUM ION;
CYCLIC GMP;
GLUTAMIC ACID;
HISTAMINE H1 RECEPTOR ANTAGONIST;
N METHYL DEXTRO ASPARTIC ACID;
N METHYL DEXTRO ASPARTIC ACID RECEPTOR;
N METHYL DEXTRO ASPARTIC ACID RECEPTOR STIMULATING AGENT;
TERFENADINE;
ANIMAL CELL;
ARTICLE;
CALCIUM CELL LEVEL;
CALCIUM TRANSPORT;
CELL CULTURE;
CELL GROWTH;
CONCENTRATION RESPONSE;
CONTROLLED STUDY;
DRUG EFFECT;
NEUROPROTECTION;
NEUROTOXICITY;
NONHUMAN;
NUCLEOTIDE METABOLISM;
PRIORITY JOURNAL;
RAT;
STEADY STATE;
ANIMALIA;
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EID: 0036205609
PISSN: 10298428
EISSN: None
Source Type: Journal
DOI: 10.1080/10298420290007583 Document Type: Article |
Times cited : (12)
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References (42)
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