Allelic gain and amplification on the long arm of chromosome 17 in anaplastic meningiomas

Brain Pathology

Volumn 12, Issue 2, 2002, Pages 145-153

  Büschges, Rainer ^a   Ichimura, Koichi ^a   Weber, Ruthild G ^b   Reifenberger, Guido ^c   Collins, V Peter ^a  

^a UNIVERSITY OF CAMBRIDGE   (United Kingdom)

^b UNIVERSITY OF HEIDELBERG   (Germany)

^c HEINRICH HEINE UNIVERSITY   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

ADOLESCENT; ADULT; AGED; ALLELE; ANAPLASTIC CARCINOMA; ARTICLE; CHILD; CHROMOSOME 17Q; CHROMOSOME ABERRATION; CLINICAL ARTICLE; COMPARATIVE GENOMIC HYBRIDIZATION; FEMALE; GENE AMPLIFICATION; GENE LOCUS; HUMAN; HUMAN TISSUE; MALE; MENINGIOMA;

EID: 0036202835     PISSN: 10156305     EISSN: None     Source Type: Journal    
DOI: 10.1111/j.1750-3639.2002.tb00429.x     Document Type: Article

References (21)

1. Increased copy number at 17q22-q24 by cgh in breast cancer is due to high-level amplification of two separate regions
2. Ps6k amplification characterizes a small subset of anaplastic meningiomas
3. Localization of PS6k to chromosomal region 17q23 and determination of its amplification in breast cancer
4. Real-time quantitative RT-PCR after laser-assisted cell picking
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6. The tumour suppressor protein NF2/merlin: The puzzle continues
7. Human glioblastomas with no alterations of the CDKN2A (p16INK4A, MTS1) and CDK4 genes have frequent mutations of the retinoblastoma gene
8. Detection of gains and losses in 18 meningiomas by comparative genomic hybridization
9. Meningiomas
10. Detection of DNA amplification in 17 primary breast carcinomas with homogeneously staining regions by a modified comparative genomic hybridization technique
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14. Four regions of allelic imbalance on 17q12-qter associated with high-grade breast tumors
15. Comparative genomic hybridization of formalin-fixed, paraffin-embedded breast tumors reveals different patterns of chromosomal gains and losses in fibroadenomas and diploid and aneuploid carcinomas
16. Evidence for the complete inactivation of the NF2 gene in the majority of sporadic meningiomas
17. Tight association of loss of merlin ex-pression with loss of heterozygosity at chromosome 22q in sporadic meningiomas
18. A novel amplification at 17q21-q23 in ovarian cancer cell lines detected by comparative genomic hybridization
19. Analysis of genomic alterations in benign, atypical, and anaplastic meningiomas: Toward a genetic model of meningioma progression
20. Quantitative analysis of neurofibromatosis type 2 gene transcripts in meningiomas supports the concept of distinct molecular variants
21. 17q23 amplifications in breast cancer involve the PAT1, RAD51C, PS6k, and SIGMA1B genes