Mitochondrial dysfunction in acute hyperammonemia

Neurochemistry International

Volumn 40, Issue 6, 2002, Pages 487-491

  Felipo, Vicente ^a   Butterworth, Roger F ^b  

^a CENTRO DE INVESTIGACIÓN PRÍNCIPE FELIPE   (Spain)

^b UNIVERSITY OF MONTREAL   (Canada)

Author keywords

Acute liver failure; Ammonia; Hyperammonemia; Mitochondrial dysfunction; Nitric oxide synthase; NMDA receptor activation; Peripheral type benzodiazepine receptors; Reactive oxygen species; Urea cycle enzymopathies

Indexed keywords

AMINE OXIDASE (FLAVIN CONTAINING) ISOENZYME B; AMMONIA; BENZODIAZEPINE RECEPTOR; BRAIN MONOAMINE; CALCIUM; N METHYL DEXTRO ASPARTIC ACID RECEPTOR; N METHYL DEXTRO ASPARTIC ACID RECEPTOR BLOCKING AGENT; NITRIC OXIDE SYNTHASE INHIBITOR; OXOGLUTARATE DEHYDROGENASE; RECEPTOR SUBTYPE; SCAVENGER;

AMMONIA BLOOD LEVEL; ARTICLE; ASTROCYTE; CALCIUM HOMEOSTASIS; CELL ENERGY; CENTRAL NERVOUS SYSTEM; CONGENITAL DISORDER; DEATH; DEGRADATION; DISORDERS OF MITOCHONDRIAL FUNCTIONS; ENERGY METABOLISM; ENZYME INHIBITION; ENZYME LOCALIZATION; HUMAN; HYPERAMMONEMIA; LIVER FAILURE; MITOCHONDRIAL MEMBRANE; MITOCHONDRION SWELLING; NEUROLOGIC DISEASE; NONHUMAN; PRIORITY JOURNAL; RECEPTOR UPREGULATION; REYE SYNDROME; SEIZURE;

ACUTE DISEASE; ANIMALS; HUMANS; HYPERAMMONEMIA; MITOCHONDRIA; NERVOUS SYSTEM; RECEPTORS, GABA-A; RECEPTORS, N-METHYL-D-ASPARTATE;

EID: 0036175623     PISSN: 01970186     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0197-0186(01)00119-X     Document Type: Article

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