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Volumn 109, Issue 3, 2002, Pages 347-355

Muscle-specific inactivation of the IGF-I receptor induces compensatory hyperplasia in skeletal muscle

Author keywords

[No Author keywords available]

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE; MYOD PROTEIN; MYOGENIC FACTOR; MYOGENIN; PROTEIN P21; SOMATOMEDIN B; SOMATOMEDIN C; SOMATOMEDIN RECEPTOR; TYROSINE KINASE RECEPTOR;

EID: 0036170286     PISSN: 00219738     EISSN: None     Source Type: Journal    
DOI: 10.1172/JCI0213503     Document Type: Article
Times cited : (124)

References (33)
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    • Wu, Z.1
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    • Correlation of terminal cell cycle arrest of skeletal muscle with induction of p21 by MyoD
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    • Halevy, O.1
  • 20
    • 0034462138 scopus 로고    scopus 로고
    • Insulin-like growth factor-mediated muscle cell survival: Central roles for Akt and cyclin-dependent kinase inhibitor p21
    • (2000) Mol. Cell. Biol. , vol.20 , pp. 8983-8995
    • Lawlor, M.A.1    Rotwein, P.2
  • 23
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    • Functional inactivation of the IGF-I and insulin receptors in skeletal muscle causes type 2 diabetes
    • (2001) Genes Dev. , vol.15 , pp. 1926-1934
    • Fernandez, A.M.1
  • 32
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    • Functional inactivation of the IGF-I receptor delays differentiation of skeletal muscle cells
    • (2000) J. Endocrinol. , vol.167 , pp. 175-182
    • Cheng, Z.Q.1


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.