Atypical protein kinase Cs are the ras effectors that mediate repression of myogenic satellite cell differentiation

Molecular and Cellular Biology

Volumn 22, Issue 4, 2002, Pages 1140-1149

  Fedorov, Yuri V ^a   Jones, Nathan C ^a   Olwin, Bradley B ^a  

^a UNIVERSITY OF COLORADO   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

12 (2 CYANOETHYL) 6,7,12,13 TETRAHYDRO 13 METHYL 5 OXOINDOLO[2,3 A]PYRROLO[3,4 C]CARBAZOLE; 2 [1 (3 DIMETHYLAMINOPROPYL) 5 METHOXY 1H INDOL 3 YL] 3 (1H INDOL 3 YL)MALEIMIDE; PROTEIN KINASE C; RAS PROTEIN;

ANIMAL CELL; ARTICLE; CELL DIFFERENTIATION; CELL LINE; CELL TRANSPORT; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME ANALYSIS; MOUSE; MUSCLE DEVELOPMENT; NONHUMAN; ONCOGENE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN TRANSPORT; SATELLITE CELL;

ANIMALS; BUTADIENES; CARBAZOLES; CELL DIFFERENTIATION; CELL DIVISION; CELL LINE; ENZYME INHIBITORS; GENES, REPORTER; IMMUNOHISTOCHEMISTRY; INDOLES; ISOENZYMES; MICE; MITOGEN-ACTIVATED PROTEIN KINASES; MUSCLE DEVELOPMENT; MUSCLE, SKELETAL; NITRILES; PROTEIN KINASE C; RAS PROTEINS; TRANSFECTION;

ANIMALIA;

EID: 0036150235     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/MCB.22.4.1140-1149.2002     Document Type: Article

References (35)

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2. Protein kinase C zeta isoform is critical for mitogenic signal transduction
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5. Retention of specific protein kinase C isozymes following chronic phorbol ester treatment in BC3H-1 myocytes
6. Overexpression of mammalian protein kinase C-zeta does not affect the growth characteristics of NIH 3T3 cells
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13. Inhibition of protein kinase C mu by various inhibitors Differentiation from protein kinase c isoenzymes
14. ERK1/2 is required for myoblast proliferation but is dispensable for muscle gene expression and cell fusion
15. Transcriptional activation of c-fos by oncogenic Ha-Ras in mouse mammary epithelial cells requires the combined activities of PKC-lambda, epsilon and zeta
16. Oncogenic Ras activation of Raf/mitogen-activated protein kinase-independent pathways is sufficient to cause tumorigenic transformation
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18. A requirement for fibroblast growth factor in regulation of skeletal muscle growth and differentiation cannot be replaced by activation of plate-let-derived growth factor signaling pathways
19. The FGF receptor-1 tyrosine kinase domain regulates myogenesis but is not sufficient to stimulate proliferation
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24. Protein kinase C and lipid signaling for sustained cellular responses
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28. Primary mouse myoblast purification, characterization, and transplantation for cell-mediated gene therapy
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34. Multiple Ras functions can contribute to mammalian cell transformation
35. MEKK1 binds directly to the c-Jun N-terminal kinases/stress-activated protein kinases