Editorial: The mystery - And importance - Of diabetic atherosclerotic vascular disease

Journal of Clinical Endocrinology and Metabolism

Volumn 87, Issue 1, 2002, Pages 33-34

  Williams, Kevin Jon ^a,b  

^a Department of Medicine   (United States)

^b THOMAS JEFFERSON UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

LIPOPROTEIN;

ATHEROSCLEROSIS; CARDIOVASCULAR DISEASE; CARDIOVASCULAR RISK; DIABETES MELLITUS; DIABETIC ANGIOPATHY; EDITORIAL; HUMAN; LIPOPROTEIN BLOOD LEVEL; PATHOGENESIS; PRIORITY JOURNAL;

CARDIOVASCULAR DISEASES; CELLS, CULTURED; DIABETES COMPLICATIONS; DIABETES MELLITUS; DIABETES MELLITUS, TYPE 2; EXTRACELLULAR MATRIX PROTEINS; FEMALE; HUMANS; LIPOPROTEINS; MALE; MUSCLE, SMOOTH, VASCULAR; PROTEOGLYCANS; RISK FACTORS;

EID: 0036146294     PISSN: 0021972X     EISSN: None     Source Type: Journal    
DOI: 10.1210/jc.87.1.33     Document Type: Editorial

References (30)

1. Diabetes mellitus and myocardial infarction: A time to act or a time to wait?
2. Improving the adverse cardiovascular prognosis of type 2 diabetes
3. Diabetes and decline in heart disease mortality in US adults
4. Risk factors for coronary heart disease in diabetics
5. Glycated haemoglobin, diabetes, and mortality in men in Norfolk cohort of European Prospective Investigation of Cancer and Nutrition (EPIC-Norfolk)
6. "Normal" blood glucose and coronary risk. Dose response effect seems consistent throughout the glycaemic continuum
7. Triglyceride-rich lipoproteins from subjects with type 2 diabetes do not demonstrate increased binding to biglycan, a vascular proteoglycan
8. The relationship between plasma and tissue lipids in human atherosclerosis
9. Initiation of atherosclerotic lesions in cholesterol-fed rabbits. II. Selective retention of LDL vs. selective increases in LDL permeability in susceptible sites of arteries
10. The response-to-retention hypothesis of early atherogenesis
11. Cellular consequences of the association of apoB lipoproteins with proteoglycans. Potential contribution to atherogenesis
12. The response-to-retention hypothesis of atherogenesis reinforced
13. Role of extracellular retention of low density lipoproteins in atherosclerosis
14. Interaction of native and modified low-density lipoproteins with extracellular matrix
15. Arterial-wall chondroitin sulfate proteoglycans: Diverse molecules with distinct roles in lipoprotein retention and atherogenesis
16. Human serum lipoproteins. II. Some effects of their intravenous injection in rats
17. Lipid accumulation in rabbit aortic intima 2 hours after bolus infusion of low density lipoprotein. A deep-etch and immunolocalization study of ultrarapidly frozen tissue
18. Comparison of apolipoprotein and proteoglycan deposits in human coronary atherosclerotic plaques: Colocalization of biglycan with apolipoproteins
19. Distribution of glycosaminoglycans in the intima of human aortas: Changes in atherosclerosis and diabetes mellitus
20. The glycosaminoglycans of the human artery and their changes in atherosclerosis
21. Relationship of sulfated glycosaminoglycans and cholesterol content in normal and arteriosclerotic human aorta
22. Cholesterol-dependent changes of glycosaminoglycan pattern in human aorta
23. Atherogenic levels of low density lipoprotein alter the permeability and composition of the endothelial barrier
24. Macrophage migration in fibrin gel matrices. II. Effects of clotting factor XIII, fibronectin, and glycosaminoglycan content on cell migration
25. Heparin, heparan sulfate, smooth muscle cells, and atherosclerosis
26. Endothelial heparan sulfate proteoglycan. I. Inhibitory effects on smooth muscle cell proliferation
27. The antiproliferative activity of arterial heparan sulfate resides in domains enriched with 2-O-sulfated uronic acid residues
28. Molecular cloning and expression of two distinct cDNA-encoding heparan sulfate proteoglycan core proteins from a rat endothelial cell line
29. Perlecan is required to inhibit thrombosis after deep vascular injury and contributes to endothelial cell-mediated inhibition of intimal hyperplasia
30. Angiotensin-II is a major mediator of diabetes-induced suppression of heparan sulfate proteoglycan assembly in key organs involved in the accelerated atherosclerosis of diabetes mellitus