Blood pressure - Independent attenuation of cardiac hypertrophy by AT1R-AS gene therapy

Hypertension

Volumn 39, Issue 5, 2002, Pages 969-975

  Pachori, Alok S ^b,d   Numan, Mohammed T ^c   Ferrario, Carlos M ^a   Diz, Debra M ^a   Raizada, Mohan K ^c   Katovich, Michael J ^b  

^a WAKE FOREST SCHOOL OF MEDICINE   (United States)

^b Department of Pharmacotherapy and Translational Research   (United States)

^c UNIVERSITY OF FLORIDA COLLEGE OF MEDICINE   (United States)

^d BRIGHAM AND WOMEN S HOSPITAL   (United States)

Author keywords

Antisense elements; Genes; Hypertension, essential; Rat, transgenic; Renin angiotensin system

Indexed keywords

ANGIOTENSIN 1 RECEPTOR; RETROVIRUS VECTOR;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ANTIHYPERTENSIVE ACTIVITY; ARTICLE; BLOOD PRESSURE; CONTROLLED STUDY; DRUG MECHANISM; FEMALE; GENE EXPRESSION; GENE THERAPY; HEART VENTRICLE HYPERTROPHY; NONHUMAN; NUCLEIC ACID PROBE; PRIORITY JOURNAL; RAT; RENIN ANGIOTENSIN ALDOSTERONE SYSTEM; SPONTANEOUSLY HYPERTENSIVE RAT;

ACTINS; ANIMALS; ANIMALS, GENETICALLY MODIFIED; ATRIAL NATRIURETIC FACTOR; BLOOD PRESSURE; CARDIOMEGALY; DNA, ANTISENSE; FEMALE; GENE EXPRESSION; GENE THERAPY; GENETIC VECTORS; MALE; RATS; RATS, SPRAGUE-DAWLEY; RECEPTOR, ANGIOTENSIN, TYPE 1; RECEPTORS, ANGIOTENSIN; RENIN; RETROVIRIDAE; RNA, MESSENGER; TREATMENT OUTCOME;

EID: 0036110088     PISSN: 0194911X     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.HYP.0000017827.63253.16     Document Type: Article

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