Increasing IKs corrects abnormal repolarization in rabbit models of acquired LQT2 and ventricular hypertrophy

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 283, Issue 2 52-2, 2002, Pages

  Xu, Xiaoping ^a   Salata, Joseph J ^a   Wang, Jixin ^a   Wu, Ying ^b   Yan, Gan Xin ^b   Liu, Tengxian ^c   Marinchak, Roger A ^c   Kowey, Peter R ^a  

^a Main Line Health Heart Center   (United States)

^b MERCK RESEARCH LABORATORIES   (United States)

^c THOMAS JEFFERSON UNIVERSITY   (United States)

Author keywords

Action potential; Arrhythmia; Early afterdepolarization; Ion channel

Indexed keywords

CALCIUM CHANNEL L TYPE; DOFETILIDE; ION CHANNEL;

ANIMAL CELL; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; HEART ELECTROPHYSIOLOGY; HEART FAILURE; HEART MUSCLE CONDUCTION DISTURBANCE; HEART MUSCLE POTENTIAL; HEART REPOLARIZATION; HEART VENTRICLE ARRHYTHMIA; HEART VENTRICLE HYPERTROPHY; LONG QT SYNDROME; MALE; NONHUMAN; PHARMACOLOGICAL STIMULATION; POTASSIUM CURRENT; PRIORITY JOURNAL; RABBIT;

EID: 0036070487     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.00076.2002     Document Type: Article

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