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Volumn 22, Issue 7, 2002, Pages 1086-1092
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NO attenuates insulin signaling and motility in aortic smooth muscle cells via protein tyrosine phosphatase 1B-mediated mechanism
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Author keywords
Atherosclerosis; Cell signaling; Growth factors; Signal transduction; Type 2 diabetes
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Indexed keywords
ADENOVIRUS VECTOR;
INSULIN;
INSULIN RECEPTOR;
INSULIN RECEPTOR SUBSTRATE 1;
MITOGEN ACTIVATED PROTEIN KINASE;
N ACETYL S NITROSOPENICILLAMINE;
NITRIC OXIDE;
PROTEIN TYROSINE PHOSPHATASE;
RECEPTOR SUBUNIT;
ANALYSIS OF VARIANCE;
ANIMAL CELL;
ANIMAL MODEL;
ARTICLE;
ATHEROSCLEROSIS;
CELL MOTILITY;
CONTROLLED STUDY;
DOWN REGULATION;
ENZYME ACTIVITY;
GENE OVEREXPRESSION;
INSULIN METABOLISM;
NEWBORN;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
SIGNAL TRANSDUCTION;
SMOOTH MUSCLE FIBER;
WESTERN BLOTTING;
ANIMALS;
ANIMALS, NEWBORN;
AORTA, THORACIC;
BUTADIENES;
CELL COMMUNICATION;
CELL MOVEMENT;
CELLS, CULTURED;
DIABETES MELLITUS, TYPE 2;
DRUG SYNERGISM;
ENZYME INHIBITORS;
FEMALE;
INSULIN;
MITOGEN-ACTIVATED PROTEIN KINASE 1;
MITOGEN-ACTIVATED PROTEIN KINASE 3;
MITOGEN-ACTIVATED PROTEIN KINASES;
MUSCLE, SMOOTH, VASCULAR;
MUTATION;
NITRIC OXIDE;
NITRIC OXIDE DONORS;
NITRILES;
PENICILLAMINE;
PHOSPHOTYROSINE;
PROTEIN-SERINE-THREONINE KINASES;
PROTEIN-TYROSINE-PHOSPHATASE;
RATS;
RATS, SPRAGUE-DAWLEY;
RECEPTOR, INSULIN;
RECOMBINANT PROTEINS;
SIGNAL TRANSDUCTION;
SRC HOMOLOGY DOMAINS;
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EID: 0036061898
PISSN: 10795642
EISSN: None
Source Type: Journal
DOI: 10.1161/01.ATV.0000020550.65963.E9 Document Type: Article |
Times cited : (45)
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References (39)
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