The inherent cellular level of reactive oxygen species: One of the mechanisms determining apoptotic susceptibility of leukemic cells to arsenic trioxide

Apoptosis

Volumn 7, Issue 3, 2002, Pages 209-215

  Togashi, K ^a,b   Masuda, H ^c   Rankinen, T ^a   Tanaka, S ^a,d   Bouchard, C ^a   Kamiya, H ^c  

^a PENNINGTON BIOMEDICAL RESEARCH CENTER   (United States)

^b MIE UNIVERSITY   (Japan)

^c MIE NATIONAL HOSPITAL   (Japan)

^d NATIONAL INSTITUTE OF HEALTH AND NUTRITION   (Japan)

Author keywords

Apoptosis; Arsenic trioxide; Reactive oxygen species

Indexed keywords

ARSENIC TRIOXIDE; REACTIVE OXYGEN METABOLITE; 2,3 DIMETHOXY 1,4 NAPHTHOQUINONE; 2,3-DIMETHOXY-1,4-NAPHTHOQUINONE; ANTINEOPLASTIC AGENT; NAPHTHOQUINONE; ORGANOARSENIC DERIVATIVE; OXIDE;

ANTINEOPLASTIC ACTIVITY; APOPTOSIS; ARTICLE; CANCER CELL CULTURE; CELL GROWTH; CELL STRAIN HL 60; CELL TYPE; CONCENTRATION RESPONSE; CONTROLLED STUDY; CORRELATION ANALYSIS; CYTOTOXICITY; FLOW CYTOMETRY; HUMAN; HUMAN CELL; LEUKEMIA CELL; PRIORITY JOURNAL; TUMOR CELL; CELL CULTURE; CELL SEPARATION; DOSE RESPONSE; DRUG EFFECT; METABOLISM; MYELOID LEUKEMIA; NICK END LABELING; PATHOLOGY; PHYSIOLOGY;

ANTINEOPLASTIC AGENTS; APOPTOSIS; ARSENICALS; CELL SEPARATION; DOSE-RESPONSE RELATIONSHIP, DRUG; FLOW CYTOMETRY; HUMAN; IN SITU NICK-END LABELING; LEUKEMIA, MYELOID; NAPHTHOQUINONES; OXIDES; REACTIVE OXYGEN SPECIES; SUPPORT, NON-U.S. GOV'T; TUMOR CELLS, CULTURED; HUMANS;

EID: 0036016917     PISSN: 13608185     EISSN: None     Source Type: Journal    
DOI: 10.1023/A:1015331229263     Document Type: Article

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