Neuronal activity determined by quantitative EEG and cortical microdialysis is increased following controlled cortical impact injury in rats

Acta Neurochirurgica, Supplement

Volumn , Issue 81, 2002, Pages 221-223

  Sakowitz, O W ^a   Unterberg, A W ^a   Stover, John F ^a  

^a CHARITÉ UNIVERSITÄTSMEDIZIN BERLIN   (Germany)

Author keywords

Anesthesia; Excitotoxicity; Glutamate; Isoflurane

Indexed keywords

CARBON DIOXIDE; GLUTAMIC ACID; OXYGEN;

ANIMAL; ARTICLE; BLOOD; BRAIN CORTEX; BRAIN INJURY; DISEASE MODEL; ELECTROENCEPHALOGRAPHY; GLUCOSE BLOOD LEVEL; HEMISPHERIC DOMINANCE; INJURY; KINETICS; MALE; METABOLISM; MICRODIALYSIS; NERVE CELL; PATHOPHYSIOLOGY; PH; PHYSIOLOGY; PRESSURE; RAT; SPRAGUE DAWLEY RAT;

ANIMALS; BLOOD GLUCOSE; BRAIN INJURIES; CARBON DIOXIDE; CEREBRAL CORTEX; DISEASE MODELS, ANIMAL; ELECTROENCEPHALOGRAPHY; FUNCTIONAL LATERALITY; GLUTAMIC ACID; HYDROGEN-ION CONCENTRATION; KINETICS; MALE; MICRODIALYSIS; NEURONS; OXYGEN; PARTIAL PRESSURE; RATS; RATS, SPRAGUE-DAWLEY;

EID: 0035989207     PISSN: 00651419     EISSN: None     Source Type: Book Series    
DOI: 10.1007/978-3-7091-6738-0_57     Document Type: Conference Paper

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7. Temporal profile of cerebrospinal fluid glutamate, interleukin-6, and tumor necrosis factor-α in relation to brain edema and contusion following controlled cortical impact injury in rats
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