Bid regulation of neuronal apoptosis

Developmental Brain Research

Volumn 128, Issue 2, 2001, Pages 187-190

  Leonard, Jeffrey R ^a   D'Sa, Cleta ^a   Cahn, B Rael ^a   Korsmeyer, Stanley J ^b   Roth, Kevin A ^a  

^a WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b HOWARD HUGHES MEDICAL INSTITUTE   (United States)

Author keywords

Bcl 2; Bcl XL; Programmed cell death

Indexed keywords

BRAIN PROTEIN; CYTARABINE; PROTEIN BID; UNCLASSIFIED DRUG;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; EMBRYO; FEMALE; IN VITRO STUDY; MOUSE; NERVE CELL; NERVE CELL DIFFERENTIATION; NERVOUS SYSTEM DEVELOPMENT; NONHUMAN; PRIORITY JOURNAL; PROTEIN DEFICIENCY; REGULATORY MECHANISM; STEM CELL; TELENCEPHALON;

ANIMALS; ANTIMETABOLITES, ANTINEOPLASTIC; APOPTOSIS; BH3 INTERACTING DOMAIN DEATH AGONIST PROTEIN; BRAIN; CARRIER PROTEINS; CASPASE 3; CASPASES; CYTARABINE; FEMALE; GANGLIA, SPINAL; GENE EXPRESSION REGULATION, DEVELOPMENTAL; IN SITU NICK-END LABELING; MICE; MICE, KNOCKOUT; MUTAGENS; NEURONS; PREGNANCY; STEM CELLS;

EID: 0035968048     PISSN: 01653806     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0165-3806(01)00174-2     Document Type: Article

References (20)

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5. L prevents this release but not tumor necrosis factor-R1/Fas death
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8. Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice
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10. Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors
11. Cell death during development of the nervous system
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18. tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c
19. Bid-deficient mice are resistant to Fas-induced hepatocellular apoptosis
20. Deficiency in caspase-9 or caspase-3 induces compensatory caspase activation