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Volumn 20, Issue 48, 2001, Pages 7085-7095

Inflammatory-type responses after exposure to ionizing radiation in vivo: A mechanism for radiation-induced bystander effects?

(5) Lorimore, Sally A a Coates, Philip J a Scobie, Gillian E a Milne, Gordon a Wright, Eric G a

a UNIVERSITY OF DUNDEE (United Kingdom)

Author keywords

Bystander; Genetics; Inflammation; Ionizing radiation; Macrophage

Indexed keywords

BETA GALACTOSIDASE; DNA; LYSOSOME ENZYME; SUPEROXIDE;

ANIMAL CELL; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; DNA DAMAGE; ENZYME ACTIVITY; GENOTOXICITY; IMMUNOHISTOCHEMISTRY; INFLAMMATION; IONIZING RADIATION; MACROPHAGE; MOUSE; NONHUMAN; PRIORITY JOURNAL; RADIATION EXPOSURE; SPLEEN CELL;

ANIMALS; APOPTOSIS; BETA-GALACTOSIDASE; BONE MARROW; BYSTANDER EFFECT; CHEMOTAXIS, LEUKOCYTE; DOSE-RESPONSE RELATIONSHIP, RADIATION; ENZYME INDUCTION; GAMMA RAYS; GENES, P53; GENETIC PREDISPOSITION TO DISEASE; GENOTYPE; INFLAMMATION; LYSOSOMES; MACROPHAGE ACTIVATION; MICE; MICE, INBRED C57BL; MICE, INBRED CBA; MICE, INBRED DBA; MICE, KNOCKOUT; NEUTROPHILS; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE TYPE II; RADIATION INJURIES, EXPERIMENTAL; RADIATION TOLERANCE; RESPIRATORY BURST; SPECIES SPECIFICITY; SPLEEN; TUMOR SUPPRESSOR PROTEIN P53; TYROSINE; WHOLE-BODY IRRADIATION;

EID: 0035950528 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204903 Document Type: Article

Times cited : (335)

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