Ionic remodeling of cardiac purkinje cells by congestive heart failure

Circulation

Volumn 104, Issue 17, 2001, Pages 2095-2100

  Han, Wei ^a,b   Chartier, Denis ^a   Li, Danshi ^a   Nattel, Stanley ^a,b  

^a MONTREAL HEART INSTITUTE   (Canada)

^b MCGILL UNIVERSITY   (Canada)

Author keywords

Antiarrhythmia agents; Electrophysiology; Heart failure; Ion channels; Remodeling

Indexed keywords

1 [2 (6 METHYL 2 PYRIDYL)ETHYL] 4 (4 METHYLSULFONYLAMINOBENZOYL)PIPERIDINE; CALCIUM CHANNEL L TYPE; CALCIUM CHANNEL T TYPE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARRHYTHMOGENESIS; ARTICLE; CALCIUM CURRENT; CELL ISOLATION; CONGESTIVE HEART FAILURE; CONTROLLED STUDY; DOG; HEART ARRHYTHMIA; HEART ELECTROPHYSIOLOGY; HEART MUSCLE FIBER MEMBRANE POTENTIAL; HEART REPOLARIZATION; HEART VENTRICLE PACING; HEART VENTRICLE REMODELING; HYPERTROPHY; NONHUMAN; POTASSIUM CURRENT; PRIORITY JOURNAL; PURKINJE CELL; SODIUM CALCIUM EXCHANGE;

EID: 0035940378     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/hc4201.097134     Document Type: Article

References (27)

1. Cellular basis of ventricular arrhythmias and abnormal automaticity in heart failure
2. Effect of vasodilator therapy on mortality in chronic congestive heart failure: Results of a Veterans Administration cooperative study
3. Sex difference in risk of torsade de pointe with d,I-sotalol
4. Repolarization abnormalities, arrhythmia and sudden death in canine tachycardia-induced cardiomyopathy
5. Electrophysiological remodeling in hypertrophy and heart failure
6. Effects of experimental heart failure on atrial cellular and ionic electrophysiology
7. Pharmacological response of quinidine induced early afterdepolarisations in canine cardiac Purkinje fibres: Insights into underlying ionic mechanisms
8. The electrophysiological mechanism of ventricular arrhythmias in the long QT syndrome: Tridimensional mapping of activation and recovery patterns
9. Purkinje-muscle reentry as a mechanism of polymorphic ventricular arrhythmias in a 3-dimensional model of the ventricles
10. Transient outward currents in subendocardial Purkinje myocytes surviving in the 24 and 48 hr infarcted heart
11. Reduced calcium currents in subendocardial Purkinje myocytes that survive in the 24- and 48-hour infarcted heart
12. + current density in arrhythmogenic subendocardial Purkinje myocytes from the infarcted heart
13. A comparison of transient outward currents in canine cardiac Purkinje cells and ventricular myocytes
14. Slow delayed rectifier current and repolarization in canine cardiac Purkinje cells
15. 2+ overload
16. 2+ currents in canine cardiac Purkinje cells
17. 2+ currents in single canine Purkinje cells
18. Simulation study of cellular electric properties in heart failure
19. Potassium channel down-regulation in heart failure
20. Ionic mechanism of action potential prolongation in ventricular myocytes from dogs with pacinginduced heart failure
21. + currents in isolated human ventricular myocytes from patients with terminal heart failure
22. Depressed transient outward current density in ventricular myocytes from cardiomyopathic Syrian hamsters of different ages
23. L-type calcium channel abundance and function with cardiac hypertrophy and failure: A review
24. Pacing-induced heart failure causes a reduction of delayed rectifier potassium currents along with decreases in calcium and transient outward currents in rabbit ventricle
25. Pause induced early afterdepolarizations in the long QT syndrome: A simulation study
26. A characterization of transient outward and delayed rectifier currents in canine atrial myocytes: Properties of currents and role of cell isolation methods
27. + currents in rabbit heart Purkinje cells