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Volumn 499, Issue 3, 2001, Pages 225-229
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Activation of p38MAPK by TGF-β in fetal rat hepatocytes requires radical oxygen production, but is dispensable for cell death
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Author keywords
Apoptosis; Hepatocyte; p38 mitogen activated protein kinase; Radical oxygen species; Transforming growth factor
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Indexed keywords
CASPASE;
CYTOCHROME C;
GLUTATHIONE;
GLUTATHIONE SYNTHESIS INHIBITOR;
MITOGEN ACTIVATED PROTEIN KINASE INHIBITOR;
OXYGEN;
PROTEIN BCL X;
PROTEIN BCL XL;
SCAVENGER;
SYNAPTOPHYSIN;
TRANSFORMING GROWTH FACTOR BETA;
UNCLASSIFIED DRUG;
ANIMAL CELL;
APOPTOSIS;
ARTICLE;
CELL DEATH;
CELL VIABILITY;
CONTROLLED STUDY;
ENZYME ACTIVATION;
ENZYME ACTIVITY;
ENZYME DEGRADATION;
ENZYME RELEASE;
ENZYME SUBSTRATE;
FETUS;
LIVER CELL;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
ANIMALS;
APOPTOSIS;
BCL-X PROTEIN;
CASPASES;
CELL SURVIVAL;
CELLS, CULTURED;
CYTOCHROME C GROUP;
ENZYME ACTIVATION;
FETUS;
HEPATOCYTES;
MITOGEN-ACTIVATED PROTEIN KINASES;
P38 MITOGEN-ACTIVATED PROTEIN KINASES;
PROTO-ONCOGENE PROTEINS C-BCL-2;
RATS;
RATS, WISTAR;
REACTIVE OXYGEN SPECIES;
TRANSFORMING GROWTH FACTOR BETA;
LETHRINIDAE;
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EID: 0035933541
PISSN: 00145793
EISSN: None
Source Type: Journal
DOI: 10.1016/S0014-5793(01)02554-6 Document Type: Article |
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Times cited : (37)
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References (36)
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