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Volumn 20, Issue 56, 2001, Pages 8142-8147
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Signal-specific and phosphorylation-dependent RelB degradation: A potential mechanism of NF-κB control
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Author keywords
I B; NF B; Phosphorylation; Proteolysis; RelB
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Indexed keywords
IMMUNOGLOBULIN ENHANCER BINDING PROTEIN;
PROTEASOME;
SERINE;
T LYMPHOCYTE RECEPTOR;
THREONINE;
TRANSCRIPTION FACTOR;
TRANSCRIPTION FACTOR RELB;
UNCLASSIFIED DRUG;
AMINO TERMINAL SEQUENCE;
ARTICLE;
CONTROLLED STUDY;
GENE EXPRESSION;
HUMAN;
HUMAN CELL;
LYMPHOID CELL;
ONCOGENE;
PRIORITY JOURNAL;
PROTEIN DEGRADATION;
PROTEIN EXPRESSION;
PROTEIN PHOSPHORYLATION;
SIGNAL TRANSDUCTION;
T LYMPHOCYTE ACTIVATION;
ANIMALS;
CELL LINE;
CELLS, CULTURED;
CYSTEINE ENDOPEPTIDASES;
HUMANS;
JURKAT CELLS;
KINETICS;
MICE;
MULTIENZYME COMPLEXES;
NF-KAPPA B;
PHOSPHORYLATION;
PROTEASOME ENDOPEPTIDASE COMPLEX;
PROTO-ONCOGENE PROTEINS;
RECEPTORS, ANTIGEN, T-CELL;
SEQUENCE DELETION;
SIGNAL TRANSDUCTION;
T-LYMPHOCYTES;
TRANSCRIPTION FACTOR RELB;
TRANSCRIPTION FACTORS;
TUMOR NECROSIS FACTOR-ALPHA;
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EID: 0035819033
PISSN: 09509232
EISSN: None
Source Type: Journal
DOI: 10.1038/sj.onc.1204884 Document Type: Article |
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Times cited : (63)
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References (23)
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