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Volumn 102, Issue 1, 2001, Pages 75-85
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Chronic nicotine exposure reduces N-methyl-D-aspartate receptor-mediated damage in the hippocampus without altering calcium accumulation or extrusion: Evidence of calbindin-D28K overexpression
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Author keywords
Calcium binding proteins; Excitotoxicity; Neuroprotection; Organotypic cultures
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Indexed keywords
CALBINDIN;
CALCIUM ION;
MECAMYLAMINE;
N METHYL DEXTRO ASPARTIC ACID;
NICOTINE;
NICOTINIC RECEPTOR;
ANIMAL TISSUE;
ARTICLE;
CELL DEATH;
CHEMOTAXIS;
CONCENTRATION RESPONSE;
CONTROLLED STUDY;
CYTOTOXICITY;
DENTATE GYRUS;
DRUG EFFECT;
DRUG EXPOSURE;
HIPPOCAMPUS;
IMMUNOREACTIVITY;
NEUROPROTECTION;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
RAT;
ANIMALS;
AUTORADIOGRAPHY;
CALCIUM;
CALCIUM RADIOISOTOPES;
CALCIUM-BINDING PROTEIN, VITAMIN D-DEPENDENT;
CELL DEATH;
DOSE-RESPONSE RELATIONSHIP, DRUG;
DRUG ADMINISTRATION SCHEDULE;
HIPPOCAMPUS;
IMMUNOHISTOCHEMISTRY;
N-METHYLASPARTATE;
NERVE DEGENERATION;
NEURONS;
NEUROPROTECTIVE AGENTS;
NEUROTOXINS;
NICOTINE;
RATS;
RATS, SPRAGUE-DAWLEY;
RECEPTORS, N-METHYL-D-ASPARTATE;
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EID: 0035793025
PISSN: 03064522
EISSN: None
Source Type: Journal
DOI: 10.1016/S0306-4522(00)00450-4 Document Type: Article |
Times cited : (52)
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References (63)
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